Jaffe Memo Was Smoking Gun of Kakistocracy’s Population and Social Engineering Schemes

GIF: WinterWatch.net/PHOTOS: Vice.com

Two dark-side Rockefeller Foundation social engineers, one Bernard Berelson and Frederick Jaffe, exchanged working ideas on the scheme of reducing U.S. fertility rates. The outlined Jaffe memo shown below was produced on March 11, 1969.

The memo, written to Berelson while he was head of the Population Council, states, “The report was prepared in behalf of Planned Parenthood’s Population Education Staff Committee as a basis for discussion of and action on the U.S. population problem by the Planned Parenthood national organization.”

Furthermore, just two years later, Berelson and Jaffe would work together on the 1972 Rockefeller Commission Report. Many of the ideas discussed in the memorandum were incorporated into the Rockefeller Report.

Read “Rockefeller Foundation: Hardly a Non-Jewish White to be Found”

The writer of the memo in 1968, Jewish social engineer Jaffe, established the Center for Family Planning Program Development, the research and public policy arm of PPFA, along with Jewish doctor Alan Frank Guttmacher, then president of PPFA. The organization was named after Guttmacher in 1974, with Jaffe as president, and spun off from Planned Parenthood in 1977.

Dr. Guttmacher (1898- 1974) founded the American Association of Planned Parenthood Physicians, now known as the Association of Reproductive Health Professionals, as a forum for physicians to discuss the birth control pill and other advances in the field. He founded the Association for the Study of Abortion in 1964. He was a member of the Association for Voluntary Sterilization. The Guttmacher Institute is named after him. In 1973, Guttmacher was one of the signers of the Humanist Manifesto II.

Not only does the premise of the Rockefeller et al social engineering scheme address population “as a problem,” it proposes draconian and downright discordian means to carry out population control. This is an infiltrative policy in so much that flying monkeys who carry out the scheme are inserted into key positions in the sistema to impose this on American society.

And the proof is in the pudding 50 years later. An examination of the elements of the proposals show clearly that it has little to do with contraception to “plan parenthood.” It is instead a full-spectrum, sinister, social-depravity agenda.

Under social constraints we see “compulsory education of children” coupled with openly “encouraging increased homosexuality.” In the current year, this is in full bloom and manifesting itself as Drag Queen Story Time.

Now comes the usual commentariat in an NBC “News” fish wrap. Ms. Marcie Bianco declares: “Women, on the other hand, are increasingly realizing not only that they don’t need heterosexuality, but that it also is often the bedrock of their global oppression.

“From Jeffrey Epstein to Miley Cyrus’s separation and Julianne Hough’s declaration that she’s ‘not straight,’” which, she says, “together have laid bare the strictures of an American patriarchy on the edge of a nervous breakdown.”

Read: Reece Committee Report from 1954 Shows Foundations Funded the Collectivist Capture of US Education

Another sinister social constraint are “fertility control agents in water.” Indeed this has been extended to feminizing males through chemicals in food as well.

ReadChemical Agents in Water and Food Supply Feminize Human Population

Person on the right is a man. Note Illuminism all seeing eye on his garb.

The herbicide atrazine is one of the most liberally applied pesticides in the world. As a result, atrazine is the most commonly detected pesticide contaminant of ground, surface and drinking water. It’s also a potent endocrine disruptor that is active at low concentrations.

National Academy of Science studies demonstrate the reproductive consequences of atrazine exposure in adult amphibians. Atrazine-exposed males were both demasculinized (chemically castrated) and completely feminized as adults. Ten percent of the exposed genetic males developed into functional females that copulated with unexposed males and produced viable eggs. Atrazine-exposed males suffered from depressed testosterone, decreased breeding gland size, demasculinized/feminized laryngeal development, suppressed mating behavior, reduced spermatogenesis and decreased fertility.

Sinister schemes center around “economic disincentives” as well. Women are encouraged to work, and then maternity leave and childcare facilities are withheld. Tax policies are altered to penalize families with children. In general the kakistocracy puts policies in place to “make life miserable” for families with children.

A most sinister entry is “chronic depression,” which obviously is designed to create less energy and desire for family raising. The other benefit of depression for the Crime Syndicate is in the (p)harma industry arm that doses millions of women and men with anti-depressives, which saps their sex drive. No, nothing to see here, move along.

Next up in these criminals’ bag of tricks is pushing compulsory abortion and sterilization. Failing in that these are heavily pushed by brainwashing and propaganda. There is a proposal to pay women to have abortions.

There is even a scheme to “discourage private home ownership.” Of course, in recent years this is defacto or shadow-instituted via heavy student debt, keeping younger people in poverty, and rolling housing bubbles and busts, which turn home ownership into a exploitative casino. In the scamdemic era, cheap money has kicked corporate ownership of rental housing into overdrive.

The Luciferians even have a plan to confine child bearing to a limited number of adults. This is negative selection as demonstrated in the movie “Idiocracy” in which only the dumbest and most inept reproduce.

The impact of mRNA vaccines on fertility has been quickly and heavily debunked by the usual suspects – even before Stage 3 studies were completed.  A simple online search reveals this. Less than a year after these injectable substances were deployed the doth-protest-too-loudly quackastocracy is already on the infertility case with all the answers and disclaimers. This is despite the fact that these substances were rushed to market as experimental products with minimal testing in humans. The impacts are not always immediate but more multi-year and may not be fully known or appreciated until it is too late.

Read: Whistleblowers, Experts Warn of Increased Risks of Infertility, Death After COVID Vaccines

Even Low Levels of Glyphosate Alter Your Gut Microbiota

low levels of glyphosate alter gut microbiota

  • Gut microbiota composition was significantly impacted when mice were exposed to glyphosate at levels approximating the U.S. Acceptable Daily Intake of 1.75 mg/kg of body weight

  • Proinflammatory T cells and Lipocalin-2, a marker of intestinal inflammation, increased after low-dose glyphosate exposure

  • Low-dose glyphosate exposure also reduced the abundance of beneficial bacteria, including Bifidobacterium pseudolongum and Lactobacillus sp in the gut

  • Low levels of glyphosate also decreased microbial short-chain fatty acid (SCFA) biosynthesis pathways, an adverse effect, since SCFAs modulate gene expression, leading to increases in beneficial anti-inflammatory regulatory T cells

  • You can reduce your exposure to glyphosate by eating organic foods; saturating your body with glycine may help provide some protection from glyphosate toxicity

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About 8.6 billion kilograms of glyphosate, equivalent to about 18.9 billion pounds, have been applied to agricultural fields and other land worldwide since 1974. The majority — up to two-thirds — was used in the last decade.

Glyphosate is a key ingredient in herbicides like Roundup, which in its earlier days was advertised as “biodegradable” and “environmentally friendly.” Monsanto even went so far as to claim it “left the soil clean” — until they were found guilty of false advertising because the chemical is actually dangerous to the environment.

It’s toxic to humans, too, and is capable of altering gut microbiota, among other health risks. Perhaps most concerning of all, given that glyphosate has been widely detected in food and water, these changes occur even at low levels of exposure.

A team of University of Iowa researchers exposed mice to glyphosate at levels approximating the U.S. Acceptable Daily Intake of 1.75 mg/kg of body weight. When their fecal samples were analyzed, they found the exposure “significantly impacts gut microbiota composition,” including altering gut homeostasis. Proinflammatory T cells and Lipocalin-2, a marker of intestinal inflammation, increased after low-dose glyphosate exposure.

Glyphosate kills plants by inhibiting the 5-enolpyruvylshikimate-3 phosphate synthase (EPSPS) enzyme. EPSPS is an enzymatic step in the shikimate pathway,

which is involved in the synthesis of the essential aromatic amino acids phenylalanine, tyrosine and tryptophan.

Since mammals do not have the shikimate pathway, it was suggested that glyphosate would not affect human health. However, some microorganisms do have the shikimate pathway, and it’s via this link that many of glyphosate’s adverse effects in humans may occur. According to the study:

“Trillions of bacteria (gut microbiota) living in the human gut play a critical role in maintaining the healthy state of the human through the regulation of several host physiological processes, including the development and maintenance of the immune, endocrine, and nervous systems.

As bacteria utilize the shikimate pathway, glyphosate could alter gut microbiota composition by inhibiting gut bacteria harboring glyphosate sensitive EPSPS enzymes. Multiple in vitro studies have verified that many gut resident microbes are sensitive to glyphosate exposure.”

In addition to increasing fecal pH levels and proinflammatory markers, the study found low-dose glyphosate exposure reduced the abundance of beneficial bacteria, including Bifidobacterium pseudolongum and Lactobacillus sp. It also decreased microbial short-chain fatty acid (SCFA) biosynthesis pathways, an adverse effect, since SCFAs modulate gene expression, leading to increases in beneficial anti-inflammatory regulatory T cells.

In short, the team explained, “We found that glyphosate exposure, at doses similar to the U.S. ADI, can alter gut microbiota composition and modulate the neuro-immune-endocrine system resulting in a proinflammatory environment.”

Stephanie Seneff, a senior research scientist at the Massachusetts Institute of Technology (MIT), has been studying glyphosate for years and has been a champion for helping to understand how and why glyphosate is so dangerous.

The increase in glyphosate usage in the U.S., as well as in Canada, is extremely well correlated with the concurrent increase in the incidence of multiple diseases, including breast cancer, pancreatic cancer, kidney cancer, thyroid cancer, liver cancer, bladder cancer and myeloid leukemia.

Research scientist Anthony Samsel is one of Seneff’s co-authors, and together they’ve suggested that one of the ways glyphosate is harmful is via disruption of glycine homeostasis. Glyphosate has a glycine molecule as part of its structure (hence the “gly” in glyphosate). Glycine is a very common amino acid your body uses to make proteins.

Samsel and Seneff believe your body can substitute glyphosate and its metabolite aminomethylphosphonic acid (AMPA) into peptides and proteins, which results in damaged peptides and proteins being produced. According to Seneff as she explains in the video above:

“I believe that in certain proteins, in certain spots, glyphosate is able to get into the protein by mistake in place of the amino acid glycine. And to understand that glyphosate is a complete glycine molecule. It’s a perfect match to glycine. Except that it has extra materials stuck onto its nitrogen atom.

… the protein that’s going to recognize glycine in order to put it into DNA has to leave the nitrogen atom outside of its pocket because the nitrogen has to hook up with the next amino acid. So the fact that the nitrogen has some stuff on it doesn’t matter to it. It says, ‘Oh, I have to fit exactly glycine very tightly.’

Glycine is the smallest amino acid. And in order to distinguish glycine from all the other amino acids all I need to do is make sure that I make a tiny space that fits only glycine …

Glyphosate will fit because it’s a perfect glycine molecule. Except the nitrogen is sticking outside of that pocket so that it could hook. So the extra stuff on nitrogen is not constrained. This is important because I think a lot of people think, ‘Oh, it can’t happen.’”

Going back to EPSPS, the bacterial version of EPSPS inserted into glyphosate-resistant Roundup Ready crop has alanine instead of glycine. But, according to Seneff, if you change the glycine into alanine by adding one extra methyl group, it ruins the protein.

“This is absolutely terrifying,” Seneff says. “They knew, ‘First we’ve got to get rid of glycine.’ And then that takes a hit on the enzyme. The enzyme doesn’t work as well because it’s got alanine there. It’s got that extra methyl group that’s in the way — the same problem that glyphosate causes.”

The arguments for why glyphosate specifically disrupts proteins that depend on glycine for phosphate binding are described more fully in a paper Seneff published together with colleagues arguing that glyphosate is a major factor in kidney failure among young agricultural workers in Central America.

Deuterium is a naturally occurring isotope of hydrogen. Provided your cell is healthy, it has deuterium-depleting enzymes and organelles that help remove deuterium from your cells. If your mitochondria are damaged by glyphosate, they’re not going to be able to eliminate the deuterium properly. This is another way glyphosate contributes to chronic disease, Seneff says.

Your cells are surrounded by structured water, which is negatively charged and contributes to your body’s energy production by supplying deuterium-depleted hydrogen to lysosomes and mitochondria. The structured water is maintained by sulfates, which makes sulfate extremely important for health.

Deuterium is everywhere, naturally, but your body has developed an intricate way to make it harmless by trapping it in the structured water, where it’s beneficial, as it actually supports the creation of structured water.

Problems arise when you cannot make enough structured water to sequester it all. Then, the deuterium gets loose, causing mitochondrial dysfunction, impairing energy production and contributing to chronic disease.

Glyphosate, however, makes sulfate dysfunctional, which in turn destroys structured water, resulting in impaired energy production in the cell.

The process is complex, but it’s important for understanding how and why glyphosate is such a pernicious and insidious toxin. Seneff explains more about deuterium and how glyphosate contributes to its buildup in your body in our video interview above.

Since at least 2014, published papers have exposed a link between glyphosate exposure and chronic kidney disease of unknown etiology (CKDu) in Sri Lankan farmers.

It’s been suggested that consumption of glyphosate-contaminated water may contribute to chronic kidney disease by facilitating the transport of heavy metals such as arsenic and cadmium into the kidneys.

In 2019, researchers again named agricultural chemicals, including glyphosate and paraquat, as possible primary factors in CKDu, noting, “[G]lyphosate causes insidious harm through its action as an amino acid analogue of glycine, and … this interferes with natural protective mechanisms against other exposures.”

A number of animal studies have linked glyphosate to liver damage as well, including one that dates back to 1979, which showed the chemical could disrupt mitochondria in rat livers.

Glyphosate is also known to trigger the production of reactive oxygen species, leading to oxidative stress. As noted in Scientific Reports, “Elevation in oxidative stress markers is detected in rat liver and kidney after subchronic exposure to GBH [glyphosate-based herbicides] at the United States permitted glyphosate concentration of 700 μg/L in drinking water.”

Researchers from King’s College London also showed an “ultra-low dose” of glyphosate-based herbicides was damaging in rats, leading to signs of non-alcoholic fatty liver disease.

People living agricultural regions, like Salinas Valley, California, may be particularly at risk.

In a study of children in Salinas Valley, exposure to glyphosate and its degradation product aminomethylphosphonic acid (AMPA) was found to increase the risk of liver and cardiometabolic disorders in early adulthood, which could trigger the development of additional diseases later in life, including liver cancer, diabetes and heart disease.

More than 80% of U.S. children and adults, ages 6 years and up, have detectable levels of glyphosate in their urine, according to data from the U.S. Centers for Disease Control and Prevention.

Out of 2,310 urine samples that were collected as part of the National Health and Nutrition Examination Survey (NHANES), 1,885 contained glyphosate levels at or above the detection limit.

Even if you don’t live near an agricultural area or use glyphosate-containing herbicides in your garden, you’re likely being exposed via contaminated food and water. Fruits, fruit juices, vegetables, oatmeal and cereals are all likely sources of glyphosate in your diet.

The use of glyphosate as a desiccant (drying agent) may be particularly problematic because it’s sprayed so near to harvest, which could result in higher residue levels and greater exposures to consumers.

In 2020, food giant Kellogg announced they’re phasing out the use of glyphosate as a desiccant by 2025,

but many other food manufacturers are still using this toxic chemical.

You can reduce your glyphosate exposure by primarily consuming organic foods. If you’re wondering what your levels are, HRI Labs has developed home test kits for both water and urine, available in my online store. I do not make a profit from the sale of these kits. I only provide them as a service of convenience.

The urine test kit reveals the amount of glyphosate you’ve been exposed to in the past two to three weeks, while the hair test shows accumulated exposure over the past three to four months.

If your levels are high, fermented foods, particularly kimchi, are potent chelators of these kinds of chemicals. Taking activated charcoal after a questionable meal can help bind and excrete chemicals as well. Remember to stay well-hydrated to facilitate the removal of toxins through your liver, kidneys and skin.

Glycine supplementation may also be a good option to help detoxify glyphosate, because to eliminate glyphosate, you need to saturate your body with glycine. Dr. Dietrich Klinghardt, who is a specialist in metal toxicity and its connection to chronic infections, recommends taking 1 teaspoon (4 grams) of glycine powder twice a day for a few weeks and then lowering the dose to one-fourth teaspoon (1 gram) twice a day.

This forces the glyphosate out of your system, allowing it to be eliminated through your urine. Considering glycine has additional benefits for longevity and disease prevention, this is a solid strategy for protection.

There are a wide variety of other uses for glycine, which is why I take about 15 grams every day, primarily with protein like eggs and beef as it rebalances the methionine to glycine ratio to decrease methionine’s negative impacts on your metabolism.

Additionally, you can use organic, grass-fed collagen, which is naturally rich in glycine. You can boost your collagen intake by making homemade bone broth using bones and connective tissue from grass-fed, organically raised animals, enjoying health benefits and helping reduce your glyphosate load at the same time.

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The information on this website is not intended to replace a one-on-one relationship with a qualified health care professional and is not intended as medical advice. It is intended as a sharing of knowledge and information from the research and experience of Dr. Mercola and his community. Dr. Mercola encourages you to make your own health care decisions based upon your research and in partnership with a qualified health care professional. The subscription fee being requested is for access to the articles and information posted on this site, and is not being paid for any individual medical advice.

If you are pregnant, nursing, taking medication, or have a medical condition, consult your health care professional before using products based on this content.

Why Watermelon Is Good for Your Cardiometabolic Health

watermelon cardiometabolic health benefits

  • Only 6.8% of U.S. adults have optimal cardiometabolic health, while an estimated 47 million have cardiometabolic disorders

  • Watermelon contains L-citrulline and L-arginine, which are nitric oxide (NO) precursors; NO helps relax blood vessels and widen arteries

  • Consuming watermelon juice attenuates reductions in heart rate variability (HRV) after high sugar consumption; low HRV is associated with increased risk of cardiovascular disease and mortality

  • Compared to people who don’t eat watermelon, those who do have healthier quality diets and increased nutrient intake

  • Among overweight or obese adults, eating watermelon led to greater satiety, including lower hunger, food consumption and desire to eat and greater fullness, compared to eating low-fat cookies

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Only 6.8% of U.S. adults have optimal cardiometabolic health,

while an estimated 47 million have cardiometabolic disorders that increase their risk of heart disease and Type 2 diabetes.

Risk factors for cardiometabolic disease include high blood pressure, abdominal obesity, elevated fasting blood sugar, dyslipidemia and elevated triglycerides.

Factors that lower these risks are therefore beneficial for cardiometabolic health. This includes lifestyle factors like eating right, exercising and maintaining a healthy weight. Specifically, in the realm of dietary strategies, watermelon has an ideal mix of nutrients to support cardiometabolic health, making it among the best food choices.

Heart rate variability (HRV) is an indicator of your body’s capacity to respond to stress. It measures the variations in time between your heartbeats — a function controlled by your autonomic nervous system (ANS).  As such, HRV is said to be a “proxy of autonomic activity” that’s associated with executive functions, emotional regulation and more.

HRV can help assess autonomic dysfunction, which, according to researchers with Louisiana State University in Baton Rouge, is an “emerging mechanism in the development of cardiometabolic disease.”

Low HRV, for instance, is associated with increased risk of cardiovascular disease and mortality. Reduced HRV is also linked to the accumulation of visceral fat, hyperglycemia, endothelial dysfunction and increased inflammation.

Watermelon contains L-citrulline and L-arginine, which are nitric oxide (NO) precursors. NO helps relax blood vessels and widen arteries. When consumed orally, l-citrulline is converted to l-arginine. These compounds show promise for heart health, in part via their effect on HRV. According to the Louisiana State University team:

“The amino acid L-arginine serves as the substrate for NO synthase to promote the enzymatic formation of NO. In healthy participants, increasing the plasma concentrations of L-arginine resulted in improved vagal control of heart rate. Other NO-promoting therapies, such as nitrates and beetroot juice, have also shown some promise in improving HRV.”

The team previously found that consuming watermelon juice for two weeks increases NO bioavailability. It’s believed that loss of NO bioavailability may play a role in reduced HRV.

They conducted a randomized, double-blind, placebo-controlled trial to determine the effects of daily watermelon juice consumption for two weeks on HRV during an oral glucose challenge (OGC), or high sugar consumption. OGC has previously been shown to reduce HRV.

The trial involved 18 participants who drank either 500 milliliters (ml) of watermelon juice or a placebo daily for the study period. It found that watermelon juice attenuated the reductions in HRV caused by OCG. According to the study:

“Using a rigorous study design, we show the efficacy of a naturally rich source of amino acids, L-citrulline, and L-arginine, to preserve HRV during a hyperglycemic episode.

These findings build on our previous work that shows WMJ supplementation protects vascular function during hyperglycemia. NO bioavailability is potentially a link between these two integrated physiological systems, but more work is required to develop a mechanistic understanding of this relationship.”

Separate research, published in the journal Nutrients, used data from the National Health and Nutrition Examination Survey (NHANES) to look into the associations between watermelon intake, nutrient intake and diet quality. Previous studies have linked watermelon extracts or supplements to a number of health benefits, including decreased pulse pressure and lower systolic and diastolic blood pressures.

However, the extracts used in the studies equate to watermelon intakes of more than 2 pounds a day.

The featured study set out to determine if eating smaller quantities of fresh, raw watermelon would also yield beneficial effects. About 98% of the study participants, which included both children and adults, consumed raw watermelon.

Among children, usual intake was 125 grams, or 5/8 cup, a day, while adults typically consumed 161 grams, or about 2/3 cup, daily.

Compared to people who didn’t eat watermelon, those who did had healthier quality diets and increased nutrient intake. The study found:

“Children and adult watermelon consumers had greater than 5% higher intake of dietary fiber, magnesium, potassium, and vitamin A as well as more than 5% lower intake of added sugars … as well as higher intake of lycopene and other carotenoids. This study suggests watermelon can increase nutrient intake as well as diet quality in both children and adult Americans.”

Even compared to other foods, such as oatmeal, mango and nuts, watermelon consumption was associated with the highest positive percent changes. For instance, eating watermelon was linked to a 3% increase in total vegetables among children and a 10% increase in adults. Protein increased 5% in children who ate watermelon, and fatty acid ratio had a 9% increase.

Watermelon is a nutrient-dense food, so it makes sense that eating it daily gives your nutrition a healthy boost. The Nutrients researchers noted, “Studies focused on raw watermelon intake are less common but have reported reduced triglycerides and LDL cholesterol, body weight, BMI, lower risk of prostate, lung, and breast cancer, as well as higher antioxidant capacity.”

Regarding the lower sugar intake among watermelon consumers, the researchers suggested, “It’s possible the sweetness of watermelon curbed the desire to have other foods with added sugars.”

A 100-gram serving of watermelon (about 1/2 cup) provides a wealth of important nutrients, including:

  • 112 milligrams (mg) potassium

  • 8.1 mg vitamin C

  • 28 µg vitamin A

  • 10 mg magnesium

  • 3 µg folate

  • 0.4 grams of dietary fiber

Watermelon is also unique in that it’s a rich source of antioxidants, including lycopene, beta-carotene, beta-cryptoxanthin, lutein and zeaxanthin.

“Watermelon has over ten and six times higher beta-carotene and beta-cryptoxanthin content, respectively, than other commonly consumed fruits,” the researchers noted. “Encouraging Americans to consume watermelon could benefit intake of certain nutrients as well as unique components with antioxidant properties.”

Diets rich in the carotenoids beta-carotene, lutein and lycopene confer greater resistance against oxidation of low-density lipoprotein (LDL) cholesterol,

which plays a role in the development of atherosclerosis. Higher plasma concentration of carotenoids was also associated with lower DNA damage.

Plasma levels of antioxidants such as lutein, zeaxanthin, vitamin E, beta-cryptoxanthin, lycopene and alpha- and beta-carotene are also inversely correlated with congestive heart failure severity.

Lycopene, a carotenoid antioxidant that gives watermelon its pink or red color, is especially noteworthy, as research suggests it may significantly reduce your risk of stroke. A study that followed men in their mid-40s to mid-50s for more than 12 years found those with the highest blood levels of lycopene were 55% less likely to have a stroke than those with the lowest levels.

L-citrulline, meanwhile, may have therapeutic usefulness for cardiovascular disease. According to a study in Cardiovascular Drug Reviews:

“Supplemental administration [of] L-arginine has been shown to be effective in improving NO production and cardiovascular function in cardiovascular diseases associated with endothelial dysfunction, such as hypertension, heart failure, atherosclerosis, diabetic vascular disease and ischemia-reperfusion injury, but the beneficial actions do not endure with chronic therapy.

Substantial intestinal and hepatic metabolism of L-arginine … makes oral delivery very ineffective … In contrast, L-citrulline is not metabolized in the intestine or liver … L-citrulline entering the kidney, vascular endothelium and other tissues can be readily converted to L-arginine, thus raising plasma and tissue levels of L-arginine and enhancing NO production.”

A 2022 meta-analysis further highlighted watermelon consumption for improvement of cardiometabolic risk factors, calling out l-citrulline intake as well:

“To conclude, longer-term l-citrulline supplementation and watermelon consumption may improve vascular function, suggesting a potential mechanism by which increased l-citrulline intake beneficially affects cardiovascular health outcomes in adults.”

Another study looked at the effects of consuming “whole blenderized watermelon,” which refers to whole watermelon, including the flesh and rind, blended together.

Overweight or obese children between the ages of 10 and 17 consumed either 1 cup of blenderized watermelon or a sugar-sweetened beverage daily for eight weeks.

Watermelon intake significantly decreased body mass index (BMI), BMI percentile, body fat percentage and HbA1c — a test that measures average blood sugar levels over three months. “Watermelon is a potential alternative to unhealthful snacks for improving anthropometry and some risk factors related to obesity in children,” the study concluded.

Watermelon, which is 91% water by weight,

also helps keep people hydrated, and this is another reason it’s so good for you. It also boosts satiety, which has benefits for weight management. In a study of 33 overweight or obese adults, participants consumed either 2 cups of watermelon or low-fat cookies daily for four weeks.

The watermelon led to greater satiety, including lower hunger, food consumption and desire to eat and greater fullness. Eating watermelon also led to significantly decreased body weight, BMI, systolic blood pressure and waist-to-hip ratio compared to cookie consumption, which led to increased blood pressure and body fat. The watermelon group also enjoyed lower oxidative stress and increased antioxidant capacity. The researchers noted:

“This study shows that reductions in body weight, body mass index (BMI), and blood pressure can be achieved through daily consumption of watermelon, which also improves some factors associated with overweight and obesity.”

A review of the evidence surrounding watermelon and l-citrulline for cardiometabolic health, spanning studies conducted from 2000 to 2020, further supported watermelon for reduced blood pressure and, potentially, weight control. Emerging evidence even suggests watermelon may boost brain and gut health by increasing NO bioavailability in all tissues.

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Disclaimer: The entire contents of this website are based upon the opinions of Dr. Mercola, unless otherwise noted. Individual articles are based upon the opinions of the respective author, who retains copyright as marked.

The information on this website is not intended to replace a one-on-one relationship with a qualified health care professional and is not intended as medical advice. It is intended as a sharing of knowledge and information from the research and experience of Dr. Mercola and his community. Dr. Mercola encourages you to make your own health care decisions based upon your research and in partnership with a qualified health care professional. The subscription fee being requested is for access to the articles and information posted on this site, and is not being paid for any individual medical advice.

If you are pregnant, nursing, taking medication, or have a medical condition, consult your health care professional before using products based on this content.

Kratom as an Alternative for Opium Withdrawal

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  • Opioid overdose is the No. 1 cause of death of Americans under the age of 50, and opioid addiction is so widespread it has led to a decline in the average life expectancy in the U.S.

  • Kratom has been used as a traditional medicine in Thailand and Malaysia for centuries, primarily to boost energy. It also has a long history of use as an opium substitute and to wean off opium

  • Kratom contains corynantheine alkaloids such as mitragynine, which interacts with opioid receptors, adrenergic receptors, serotonin receptors, dopamine receptors and adenosine receptors

  • While the alkaloids in kratom bind to opioid receptors, animal trials suggest the abuse potential of kratom is very low

  • A compound found in dried kratom leaves, 7-hydroxymitragynine is known to be a fast-acting full opioid agonist that substitutes directly for morphine and can be habit-forming. This compound does not appear to be present in detectable amounts in fresh extracts, so using tea from fresh leaves and/or extracts may be a way to circumvent its addiction potential

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Editor’s Note: This article is a reprint. It was originally published June 24, 2018.

With opioid overdose being the No. 1 cause of death of Americans under the age of 50, it’s quite clear we need safer alternatives to pain management. Rampant opioid addiction is so significant it has even led to a decline in life expectancy in the U.S.

In this interview, Christopher McCurdy, professor of medicinal chemistry at the University of Florida College of Pharmacy and former postdoctoral fellow in opioid chemistry at the University of Minnesota under a National Institutes of Health (NIH) postdoctoral training fellowship, discusses the use of kratom for pain relief and opioid withdrawal.

He’s one of just a handful of American scientists studying kratom, which he’s been investigating for nearly 15 years. According to McCurdy:

“[A]bout 115 or 116 people a day [are] now dying from opioid overdose … It’s surpassed car accidents and other forms of death … This plant, I believe, has real potential to help curb this. It may not be the single solution to the crisis, but it certainly could help.”

Kratom (mitragyna speciosa) is a native tree of Thailand and Malaysia, but it grows all over Southeast Asia in tropical areas. While part of the coffee family, it has a very different chemistry than coffee beans. Kratom has been used in traditional medicine in Thailand and Malaysia for centuries, primarily to boost energy. It also has a long history of use as an opium substitute.

“A lot of opium smokers who would run out of opium would use kratom preparations to tide them over until they could get their opium. They also found that it was a very good way to wean themselves off of opium or wean other family members or friends from opium,” McCurdy says.

“They would … pluck the leaves fresh in the morning, brew a tea and then drink that tea two or three times a day … That brings about a lot of questions scientifically, as to why does that work, and is it habit-forming and habituating itself?”

Unfortunately, there’s not much research being done on kratom, in part because you cannot patent natural products such as plant material. You also cannot patent the plant’s use for opioid withdrawal or pain relief of antidepressive treatment, as all of these therapeutic potentials are covered under the historical use of kratom. McCurdy explains:

“If you were to produce any of the molecules through a patentable process, like a total synthesis or an enzymatic or biochemical synthesis, then that could be a product that could be patented. That process could be patented. That could move forward into commercialization. But simply getting the plant material itself as it’s available today in the marketplace and making an extract … does not lend itself to a patentable standpoint …”

McCurdy has studied how kratom affects opiate addiction and withdrawal. The plant contains a number of alkaloids (nitrogen-containing compounds). Morphine is an example of an alkaloid found in the poppy plant. Kratom contains corynantheine alkaloids, the primary one being mitragynine, thought to be responsible for most of its pharmacological effects. That said, there are many other alkaloids in the plant, and many of its compounds work synergistically to cause beneficial effects.

Mitragynine does have opioid activity. It and many other alkaloids in the kratom plant were recently called out as opioids by the Food and Drug Administration (FDA) commissioner. “A lot of people were upset about that at first, but I think they need to understand that an opioid is any molecule that can interact with opioid receptors or those proteins in the body,” McCurdy says.

In other words, an opioid is not identical to an opiate, derived from opium poppy, such as morphine, oxycodone or oxymorphone. Opioid is a generic term that include even endogenous endorphins that bind to opioid receptors in your body.

While it has opioid activity, the mitragynine molecule is different from other opioid molecules. McCurdy explains:

“We initially sent out purified alkaloid of mitragynine for a screen across a whole panel of central nervous system drug targets … What we found was a really remarkable profile of this molecule. For example, morphine … is pretty selective for opioid receptors. Mitragynine binds with opioid receptors … but it also interacts with adrenergic receptors, serotonin receptors, dopamine receptors, and adenosine receptors.

Adenosine receptors are the target for caffeine. It kind of explains why some of these alkaloids in the plant might cause this excitation or stimulant-like effect. It also interacts with alpha-2 adrenergic receptors, [which] are … used in opioid withdrawal. Agents that activate alpha-2 receptors, like clonidine, are used in opioid withdrawal treatment to stop withdrawal symptoms such as shaking, sweating and heart racing …

In all honesty, when I got the report back from the company that screened the molecule, I thought, ‘Wow. We just found nature’s answer to opiate addiction,’ because here it was interacting with many of the same targets that we would target pharmacologically on an individual basis …

Then we went on to pursue much more detailed pharmacology studies, looking at what is it doing in animals addicted to morphine and how the animals behave when we take away their morphine and turn them over to mitragynine.

What we saw was really remarkable. We compared mitragynine to methadone and buprenorphine, the two marketed drugs to treat opioid addiction and opioid withdrawal. What we found was a much cleaner profile. [Kratom] wasn’t incredibly superior … but it seemed to be milder.

It activates opioid receptors. So does methadone and buprenorphine, but buprenorphine and methadone seem to be full agonists or activators of opioid receptors, whereas mitragynine, we think, is a partial agonist …

Also, it activates a different signaling pathway once the receptor is turned on … It can be thought of as different inputs into a television … where the system is on but we’re getting input from a Blu-ray player or we’re getting input from a satellite — just different ways of signaling.

Mitragynine seems to signal in a way that seems to not cause respiratory depression, at least to the extent that other traditional opiate drugs do … Respiratory depression or stopping breathing is essentially the cause of death from opiate overdose. Opioids are pretty safe to your body’s organs, except for the fact that they are central nervous system-depressants. They will shut everything off.”

As a therapeutic agent to help people get off opioids, kratom offers significant benefits. In the CNN news report above, Dr. Sanjay Gupta talks to two opioid addicts who kicked their habit with kratom, and interviews McCurdy about kratom’s superior ability to serve as a treatment for opioid addiction.

It does, however, need to be regulated, McCurdy believes, to avoid the hazardous mixing with other far more dangerous drugs. It is this mixing, he claims, that is responsible for the deaths that have been linked to kratom.

So, just how does kratom work to curb opiate addiction? As explained by McCurdy, there are three traditional opioid receptors: mu, delta and kappa, all three of which are associated with numbing or dulling pain. In other words, they’re analgesic receptors. They block or slow pain signal transmissions at the spinal cord level, so your brain doesn’t process the pain signals as much.

Mu was named for its ability to interact with morphine. The mu receptor is responsible for the euphoric effects associated with opiates. It’s also primarily responsible for respiratory depression. The delta receptor is also a target for selective analgesics, and does not appear to have as strongly addictive capabilities as the mu receptor.

Unfortunately, the delta receptor is linked to convulsions, and many drug trials aimed at the delta-selective opioid receptor had to be halted due to seizures that could not be resolved. Kratom does not appear to significantly interact with delta receptors.

The kappa opioid receptor, while good for killing pain, causes dysphoria — aversive-type feelings. “In the ’70s and ’80s, they thought they discovered a selective kappa opioid non-addictive painkiller that was just as potent as morphine, if not more potent,” McCurdy says.

But when the human clinical trials began, the drug failed. “The people who took the drug said, ‘I don’t know what that was, but don’t ever give it to me again.’ They felt so awful they dropped out of the trials even after a single dose.”

Kratom appears to be a partial agonist for all of these receptors, only weakly affecting delta and kappa. But, if the mu receptor is the primary target, doesn’t that mean kratom is addictive? At present, animal trials suggest the abuse potential of kratom is actually quite low. McCurdy explains:

“I have a paper under review right now in addiction biology with a colleague of mine, Scott Hemby, from High Point University in North Carolina … [looking] at what the actual abuse potential is for mitragynine. He trained rats to self-administer morphine.

They were able to learn to press a lever and they would get injections of morphine … Once you’ve trained those animals, you can then substitute that morphine for some other drug and see if they think it’s like morphine, right?

We substituted mitragynine and they stopped self-administering. It seemed like, ‘Well, this is interesting. Maybe it doesn’t activate the mu opioid receptor in an addictive manner or in a manner that would produce addiction.’ Of course, … we did it in a limited fashion at the moment because we’re hoping to get some grant funding to really pursue this.

But the fact of the matter is many drugs will substitute for morphine … like oxycodone or oxymorphone, that we know are used clinically. But mitragynine did not substitute. What was even more interesting is we couldn’t train the animals to self-administer mitragynine to themselves over several doses. This was a very strong indicator that mitragynine doesn’t have an abuse potential. In fact, that’s kind of the sort of story of our paper.”

Another compound in some processed and dried kratom products, 7-hydroxymitragynine, is known to be a full opioid agonist, and a very fast-acting and powerful one that does substitute directly for morphine and can be habit-forming. It does not appear to cause severe respiratory depression, however.

This compound is not present in fresh extracts, and McCurdy’s team is trying to determine exactly how, when and why the compound is created. It could be an oxidative byproduct created during the drying process, or it could be the result of different growing regions, microclimates, the presence of certain insects or soil microbes, for example.

“I think what’s even more interesting about this study we did — we treated animals with mitragynine that were already addicted to morphine, and then re-exposed them to morphine self-administration … It decreased their intake of morphine. This was pretty exciting, because it also shows that there’s some therapeutic potential in reducing opioid intake.

That begs the question about if there is 7-hydroxy present in a very small amount, and you have maybe 20 times or more of mitragynine relative to the 7-hydroxy. Is that actually counteracting the effect of the 7-hydroxy compound so it’s not as bad? It’s really hard to say at this point, because we don’t have all the science done.

We’ve got lots of additional studies planned and hope we can figure out what’s going on there. But our take-home is that kratom, as a whole, is pretty much on the addictive level of coffee. It’s really not that harsh of an addictive plant, but it depends. It’s starting to look like it may depend on what level of this 7-hydroxymitragynine is present in the material or in the product that’s utilized.”

Right now, the evidence is leaning toward 7-hydroxymitragynine being the result of oxidative stresses from drying the leaves, which suggests using fresh leaves to make tea, or taking an extract, would eliminate the risk associated with this oxidative metabolite, essentially eliminating the addictive potential of kratom. “That’s our hypothesis right now,” McCurdy says.

“We’ve been fortunate to gain collaborations with the University of Science, Malaysia, where they have actual cultivars of Mitragyna speciosa trees that they can use and study.

We’ve been studying fresh extract material from them under an agreement.” McCurdy’s team is also investigating traditional preparations made by users in Malaysia, and have received samples from trees grown in the native environment. None of the fresh samples have 7-hydroxymitragynine in detectable amounts.

McCurdy has created a modified kratom tea, in which certain alkaloids and plant chemicals were removed, thereby eliminating all side effects. Once perfected, this would be a patentable product that could be used therapeutically to counteract opioid withdrawal symptoms without causing any. One obstacle that needs to be overcome is finding a verifiable kratom source that meets FDA standards to ensure public safety.

“Basically, if it’s a clean, good product that we can get on a regular basis and be able to standardize, that would be the next thing. You don’t want to have the product being really good one day and not so good the other because the alkaloid content has changed from one batch to the other. You have to have batch-to-batch consistency,” he says.

To solve this issue, he’s working with horticulturists at the University of Florida to understand the conditions necessary to allow kratom to grow in the U.S., particularly Florida. Could they be grown in a greenhouse? What types of nutrients do they need? He’s also been approached by commercial growers looking into the feasibility of growing kratom trees.

“I think there’s an interest in it. It may be a groundswell of interest to really see if we can create a new type of product for these growers to look at. That product would ensure us as researchers or producers of a pharmaceutical that we would have a good reliable source that we can control and understand,” he says.

“We could then hopefully blend up and produce a really top-notch product that we can do controlled clinical trials with, and then move that on to helping people. That’s the whole goal here. It’s to really get help to a lot of these opiate addicts and hopefully get them off opiates.”

Kratom is legal in most states, but not all. To check the legal status for your state, see Speciosa.org’s legality map.

In Florida, kratom is legal in all areas except Sarasota, where kratom was banned. In the fall of 2016, the U.S. Drug Enforcement Agency (DEA) sought to add mitragynine, 7-hydroxymitragynine and kratom to the Schedule 1 of the Controlled Substances Act.

Schedule 1 means the substance has absolutely no medical use and is highly addictive. This classification essentially halts all research on the substance.

There was no science or evidence to back up the DEA’s rationale, and the agency was contacted by a number of researchers expressing their objections. Researchers even contacted Congress for congressional intervention.

“Suffice it to say, there were many points of pressure on to the DEA to not make this scheduling official,” McCurdy says. “The DEA decided to open up a 30-day comment period to obtain information. Scientists and people who have benefited from it flooded the DEA with messages.”

The DEA received more than 23,000 individual comments urging them not to add kratom to Schedule 1. The opposition worked, and the DEA dropped it. This was largely a result of Chris Bell telling the story on Joe Rogan’s podcast and encouraging their viewers to write in. Rogan’s podcast has tremendous influence as this is the first time the DEA has overturned an action like this. I’ve included this historic podcast below for your convenience.

chris bell

Still, the threat is there. They could, at any time, decide to make it illegal. “We had to actually shut down our research while that threat was underway, because I didn’t want to wake up one morning, come into work and be in violation of federal law at the university. I don’t have a Schedule 1 research license,” McCurdy says.

According to McCurdy, kratom has a “remarkable” safety profile. While there have been a few reported deaths associated with kratom in the U.S., most have been in combinations with other drugs.

A couple of cases have reported only finding kratom or mitragynine alkaloids in the deceased person’s system, but in McCurdy’s view, it’s a tough sell that kratom alone would be responsible, as this herb has been used for hundreds of years in Malaysia and Thailand without any history of lethal consequences.

“We just don’t know enough about how it’s being metabolized,” he says. “If somebody has some impaired function; they may be on a high-protein diet or they may be on some other type of diet that they think is healthy, but unfortunately isn’t healthy when taking kratom. We don’t know the answers to all of these questions. There’s a lot more work that has to be done.

However, in the indigenous population where this is used, in Malaysia and in Thailand, there haven’t been any reports in the history associated to only kratom. In fact, it’s never been a drug of abuse or a drug that’s sought out for pleasure. It’s a societal norm. It’s just like [what] coffee is in the United States. [They] drink it in the morning before they go to work. They drink it in the mid-afternoon …

Many of the men will gather in the evenings and drink a glass or two of kratom tea as they socialize … It’s been safe for hundreds of years … Any death is a sad event. We don’t like to see it. But we need to understand what’s going on and how it’s happening, even in the few cases that have been reported.

Is it a combination with antidepressants that’s a problem? Is it a combination with other drugs, like muscle relaxants? We just don’t know enough of these answers yet to really tell people how to safely use the product, although millions of consumers are using it and appear to be using it safely.

The other issue on safety comes back to … getting a reliable source of the biomass … I’ve heard rumors that some products have been shipped into the United States under the form of kelp, so just as seaweed. It comes in wet and damp. That could be some of the problems that we’re seeing now with the salmonella outbreak that has been linked to kratom.”

There are four different types of kratom: red, white, green and Maeng Da. While the white and green are considered the same (the only difference is the timing of when the leaves are picked), the red-veined version is different. Many claim the red-veined leaves have more alkaloids and the plant is a more potent version, but based on McCurdy’s testing, the potency has less to do with the vein or strain and more to do with location.

“We’ve analyzed many products that are available in the marketplace through ultra-high performance liquid chromatography and mass spectrometry. What we’ve learned from that is sometimes the ones that people think are the strongest — the red vein being — actually have the lowest [alkaloid] content compared to the products that are cheaper.

But, again, it’s one of these things where there are no real labeling laws. There’s no consistency. You could buy the red vein one day, buy it the next week and it’s less potent. Go back in the next week and the potency’s back up … But from what we’ve seen in the natural environments, the difference is really going to be the microclimates …

The Maeng Da strain, touted as one of the high-end and superior strains, comes from a horn-shaped leaf called tooth leaves. Instead of a nice smooth leaf, like what you would see on mitragyna leaf, you see it’s smooth at the bottom and then real jagged edges up to a point. Again, we haven’t seen a huge difference in any of these supposed strains. A lot of it comes down to marketing, and what can play to the consumer environment.”

Traditionally, kratom has been used as a water extract and as tea made from the leaves. McCurdy has done a systematic study to evaluate various solvents, including ethanol, methanol and acetone, to determine the best extraction method. He’s also studied the various extracts in animals, finding there are definitive differences between them. Extracts of bark, stems, roots and flowers have also been investigated.

“We do know there’s a higher concentration of alkaloids in the vein than there is in the flesh of the leaf material,” he says. “Most of the products that we see coming into the U.S. are whole leaf, just chopped up or ground up, so they do have the vein … One other thing I find really fascinating is the bark or the branches. If people feel they need a boost, they will put the bark branches into their tea as they brew it.

They say it makes it much stronger. We really haven’t found out what that molecule is that’s helping to make it stronger yet … We haven’t seen much of a difference outside of the leaves. There are different compounds, of course … that are in higher quantities in the stems [and] fruits. We’re trying to pursue and see what the pharmacology is that’s associated to them …

Most of the time that people brew teas from the leaf material, they’ll also add citric acid, lemon juice or orange juice to the water to make it more acidic and pull more of those compounds out. I think, in theory, it should be a very similar process to the water extract. But the water extract will concentrate it better.”

While kratom appears to have a favorable safety profile compared to opioids, this isn’t to say that kratom usage is without risk and can be used without concern. It’s important to recognize that kratom is a psychoactive substance and should not be used carelessly. There’s still very little research showing how to use it safely and effectively, and it may have a very different effect from one person to the next.

Also, while it may be useful for weaning off opioids, certain types of kratom could potentially be addictive. So please, do your research before trying it. One place to start is the American Kratom Association.

Also know there are many other safe and effective alternatives to prescription and over-the-counter painkillers. If you’re looking for safer options for pain relief than opioid drugs, please see these options for treating pain without drugs.

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Uber Eats to deploy an additional 2,000 delivery robots, putting more jobs in peril

Image: Uber Eats to deploy an additional 2,000 delivery robots, putting more jobs in peril

(Natural News) Food delivery company Uber Eats has announced that it will expand the number of four-wheeled robots that will be delivering food on the company’s app.

The robots are made by Serve Robotics, and the decision to expand the use of robots was made after a successful pilot of the robot delivery program in Los Angeles. The pilot program, which began in the West Hollywood neighborhood last May, was wildly successful.

The robots serve customers from 10 a.m. to 9 p.m. every day, and Uber Eats claimed the use of robot deliveries in the city grew by more than 30 percent month-over-month and more than 200 restaurants in the city have already signed up to offer deliveries using the small robots. (Related: Generative AI could replace up to 300 million mostly white-collar jobs worldwide.)

Serve Robotics and Uber Eats will now be expanding the program to several other markets in the United States.

“This partnership is a major step towards mass commercialization of robotics for autonomous delivery, and it is a testament to the success of our partnership,” said Serve Robotics CEO Ali Kashani in a statement. “We are excited to continue our work with Uber to bring this innovative technology to more cities across the country.”

Serve Robotics’ delivery robots are about the size of a small shopping cart. They are about waist-high with four wheels and a payload dimension that could easily fit two large shopping bags or four large pizza boxes. They can carry up to 50 pounds of food or items and they use the sidewalks in the cities they operate in to conduct their deliveries.


The delivery robots are electric powered and use artificial intelligence and several cameras and sensors to help them navigate their surroundings. The AI detects when other people or cars are in its path and the robots know when to avoid collisions.

A customer who places a food order on the Uber Eats app gets asked whether they prefer to receive their items via a sidewalk delivery robot or a human courier, with the change not costing the customer anything.

The robot travels up to seven miles per hour on the sidewalk and has a delivery range of about a mile. Kashani claimed customers usually receive their items within 30 minutes of placing their orders.

Upon arrival at the customer’s location, the robot sends a message that it has arrived along with a unique code to input to open the payload lid, ensuring that only the customer can retrieve the order.

Use of delivery robots to expand to rest of California, Texas and Canada

The partnership is slated to deploy up to 2,000 delivery bots through the beginning of 2026. Once completed, this deal between Serve Robotics and Uber Eats would be one of the largest deployments of robotic delivery fleets ever in the United States.

“We expect our rapid growth on Uber Eats to continue,” said Kashani. “We expect to operate an increasing number of [robots] on Uber Eats as our coverage and delivery volume on Uber increases.”

Serve Robotics currently operates a fleet of about 100 robots in Los Angeles. With its partnership with Uber Eats, the company plans to deliver in other cities in California, Texas and even Canada.

“It’s been an incredible experience when you’re pioneering something like this to a city,” said Kashani. “The team and I right now are in the right time and the right place to be working on this and part of history. We are learning how to make robots friendly and likable while cutting down on traffic congestion.”

In addition, Serve Robotics already uses its fleet to conduct deliveries for the convenience store chain 7-Eleven in Los Angeles. It is already working with other companies to expand the use of robots for deliveries in other areas, such as Vancouver for Pizza Hut and Walmart for the entire state of Arkansas.

Serve Robotics is even working with self-driving car company Motional to trial delivering food in Santa Monica using the company’s self-driving Hyundai Ioniq 5 battery-electric cars.

Uber Eats has also been testing different delivery robot options in other locations. It launched a partnership with autonomous delivery robot company Cartken to test the robots in Miami, and following a successful trial expanded that partnership to several parts of Virginia.

Learn more about the takeover of human jobs by robots and artificial intelligence at Robotics.news.

Watch this video from “Future Now” on AMP News discussing the massive job losses caused by automation.

This video is from the AMP News channel on Brighteon.com.

More related stories:

IBM to replace 8,000 workers with AI automation; no more new hires for roles that can be filled by AI.

Walmart converting 65% of its stores to “automation” – human employees will be let go.

ROBOCROPPERS: John Deere planning to replace farmers with fully automated farming vehicles by 2030.

Walmart announces expansion of drone deliveries to 4 million households in 6 states.

Grubhub, Yandex team up to use robots to deliver food on college campuses.

Sources include:









SHRINKING BIRD: Twitter now worth just one-third of what Elon Musk paid to purchase it

Image: SHRINKING BIRD: Twitter now worth just one-third of what Elon Musk paid to purchase it

(Natural News) Twitter is now valued at just about one-third of what owner Elon Musk paid for it last year, according to a financial services company.

Billionaire and tech giant Musk took over the social media platform in October 2022 by paying a whopping $44 billion for the company, including $33.5 billion paid in equity. Now, Boston-based financial services company Fidelity Investments noted that the company’s value has shrunk by around 66 percent since then. (Related: Elon Musk steps down as Twitter CEO, taps WEF globalist vaccine pusher Linda Yaccarino as replacement.)

Fidelity, under its Fidelity Blue Chip Growth Fund, was one of a group of outside investors that helped Musk finance his takeover of Twitter. The company’s stake in Twitter, which now exists under Musk’s X Holdings Corporation, was valued at $19.7 million in October at the time Musk took ownership of the social media platform.

Since then, Fidelity has repeatedly marked down the value of its stake in the social media platform. In November, the stake’s value was reported to have shrunk massively to around $8.63 million. By Jan. 31, the fund’s value had shrunk to $7.8 million. And as of April 28, the holding was now valued at nearly $6.55 million.

It is not entirely clear how Fidelity arrived at the new, lower valuation of its stake on Twitter or whether it receives any non-public information from the company. But Musk himself has admitted recently that he believes Twitter is now worth less than half what he paid for it.

In a leaked email, Musk stated that he believes Twitter is now worth less than $20 billion, which tracks with Fidelity’s estimated value of the company at around $14 to $15 billion.


Despite this, Musk believes the social media platform still has “incredible potential.”

“Although obviously, myself and the other investors are obviously overpaying for it with Twitter right now,” he said.

Dan Primack, business editor at Axios, further warned that Fidelity’s mark down of Twitter shares is on a one-month lag, and it will be interesting to see how Musk’s decision to replace himself as the platform’s chief executive officer will impact the company’s valuation. The result of this turnover won’t be seen until the end of June once Fidelity and other investment companies that have stakes in Twitter release their reports.

Twitter has been dealing with financial issues since Musk takeover

Twitter has been struggling financially ever since Musk took over, with the transition to his ownership saddling the company with over $13 billion of debt.

Musk’s own erratic decision-making and other challenges have led to advertising revenue falling by more than 50 percent. In 2021, a year before Musk’s takeover, more than 90 percent of Twitter’s revenue came from advertising.

In the last two months alone, the top 50 advertisers on Twitter only spent around $83 million on the platform, down from the more than $102 million they spent in the same period last year.

“Many advertisers don’t trust him based on past behavior or don’t want to be associated with him,” said market research company Insider Intelligence Principal Analyst Jasmine Enberg.

An attempt to recoup some of this lost revenue by selling subscriptions through the Twitter Blue program has so far failed to take off. At the end of March, less than one percent of Twitter’s monthly active users have signed up for the program.

Earlier this month, Musk named former NBCUniversal advertising chief Linda Yaccarino as Twitter’s new CEO as the company struggles to reverse a massive slump in advertising revenue and navigate an overhaul that has turned the company’s ability to operate and generate revenue upside down.

Musk’s own investment in the social media platform is now worth less than $9 billion, according to the Bloomberg Billionaires Index, which used Fidelity’s own valuation to calculate the value of his holding.

The latest markdown from Fidelity has erased about $850 million from Musk’s own massive $187 billion fortune – which is up more than $48 billion this year, thanks to a 63 percent surge in Tesla’s share price.

Find the latest news about Elon Musk and Twitter at ElonMuskWatch.com.

Watch this short video exposing the truth behind Twitter’s new CEO, Linda Yaccarino, an active member of the World Economic Forum.

This video is from the ThriveTime Show channel on Brighteon.com.

More related stories:

After cooperating with Feds at Twitter, Jack Dorsey invokes JFK call to destroy CIA, FBI, NASA.

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BAD ELON: New Twitter CEO is a vaccine mandate globalist and lockdown pusher – she will NEVER support free speech.

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Sources include: