By Anna Von Reitz
By Anna Von Reitz
It has been 13 years since the U.S. Food and Drug Administration (FDA) supplied fast-tracked approval for Merck’s Gardasil vaccine—promoted for the prevention of cervical cancer and other conditions attributed to four types of human papillomavirus (HPV). The agency initially licensed Gardasil solely for 9- to 26-year-old girls and women, but subsequent FDA decisions now enable Merck to market Gardasil’s successor—the nine-valent Gardasil 9 vaccine—to a much broader age range—9 to 45 years—and to both males and females.
As a result of Gardasil’s expanding markets not just in the U.S. but internationally, the blockbuster HPV vaccine has become Merck’s third highest-grossing product, bringing in annual global revenues of about $2.3 billion. However, Gardasil’s safety record has been nothing short of disastrous. Children’s Health Defense and Robert F. Kennedy, Jr. have just produced a video detailing the many problems with the development and safety of Gardasil. Please watch and share this video so that you and others may understand why Mr. Kennedy refers to Merck’s methodologies as “fraudulent flimflams.”
What follow are 25 key facts about Gardasil/Gardasil 9, including facts about the HPV vaccines’ clinical trials and adverse outcomes observed ever since Merck, public health officials and legislators aggressively foisted the vaccines on an unsuspecting public.
As suggested in the conclusion to the 2018 book The HPV Vaccine on Trial, the rollout of Gardasil in 125 countries worldwide has illustrated—in an all-too-real and shocking manner—the phenomenon that prompted Hans Christian Andersen to write “The Emperor’s New Clothes.” Around the world, over 100,000 Gardasil-related adverse events have now been reported to the FDA and WHO, and accounts continue to multiply of “scandal, lawsuits, severe injuries, and deaths.” For almost 200 years, Andersen’s story has taught readers about the need to speak the truth, pay attention to evidence and listen to children. The rosy narrative manufactured for the dangerous Gardasil vaccine must not be allowed to hold sway any longer. It is time, in the words of the HPV Vaccine on Trial authors, to proclaim—loudly—that “the Emperor has no clothes.”
St. John’s wort (Hypericum perforatum) is named after St. John the Baptist, since it’s usually in full bloom by June 24, the saint’s feast day.1 The plant bears yellow flowers2 with oblong petals3 that have black dots on their edges. It can stand 1 to 3 feet tall, and has multiple reddish stems4 and yellow-green leaves.5 Although primarily found in Europe, St. John’s wort can now be found growing in the U.S.,6 China, northern Africa, western7 and eastern Asia, Australia and New Zealand.8
The plant typically blooms during summertime, and can be seen growing in woods, hedges, roadsides, pastures and meadows.9 St. John’s wort also bears sticky fruits with three-chambered capsules10 that hold multiple small, black and pepper-like seeds11 with a resinous smell.12
Under certain conditions, St. John’s wort may be helpful for your mental health if you’re feeling depressed — provided you aren’t currently taking prescription antidepressants, as the combination of the two could cause serious increases in your serotonin levels.13
Used alone and under the supervision of your health care provider, the herb contains compounds such as hypericin, hyperforin, flavonoids and flavonoid derivatives, xanthone derivatives, amentoflavone, biapigenin and volatile oils that may help combat depression.14,15
An analysis of 35 studies published in Systematic Reviews in 2016 revealed that St. John’s wort assisted in alleviating depression symptoms better than a placebo, and similarly to typical conventional drugs.16
Authors of a 1997 Pharmacopsychiatry animal study discovered that St. John’s wort extract may work like antidepressants by helping prevent reuptake of the neurotransmitters serotonin, dopamine and norepinephrine, and promoting positive effects in mice who were given the substance.17
This herb may also help address mental health problems such as anxiety or seasonal affective disorder (SAD),18 which normally occurs during the winter months because of a lack of sunlight.19 Aside from impacts toward your mental health, St. John’s wort may also help:
St. John’s wort was used to treat wounded soldiers during the Crusades,32 and drive out the “inner devil” in people during Medieval times. However, St. John’s wort’s history of medicinal use dates back to ancient Greece, where it was believed to possess “magical powers.”33 It was also recommended to be used against hallucinations34 by Paracelsus, a Swiss man considered “the father of chemistry and the reformer of material medica” (among many other nicknames).35
The use of St. John’s wort is prevalent in some European countries, such as Germany, against mental health issues like depression.36 St. John’s wort can be found in other forms such as tinctures, oils or balms that may help promote healing of burns, wounds, insect bites and bruises.37,38 You may also encounter St. John’s wort products such as supplements,39 liquids, powders40 and teas.41,42
St. John’s wort leaves and flowers can be used to make tea43 that may assist in:44
Remember that if you have high blood pressure levels, are taking antihypertensive50 or antidepressant medications, or would need to undergo surgery, drinking St. John’s wort tea may lead to adverse effects.51 Also, do not take St. John’s wort without consulting a health care practitioner if you are on prescription medications of any sort including birth control pills.52
Are you interested in trying St. John’s wort oil? Initial research has revealed that it possesses anti-inflammatory capabilities,53 and may help:
If you plan on using this oil, consult a physician and take an allergen patch test to check for potential side effects. Dilute the essential oil with a carrier oil like sweet almond, olive, jojoba or coconut, or add it to other oil blends, since it can deliver a warm, balsamic, herbaceous and sweet scent. St. John’s wort oil blends well with clary sage, cedar,59 lavender and lemon balm oils.60
No matter what form of St. John’s wort you use, remember that it may lead to photosensitivity61 or sun sensitivity.62 If you’re using St. John’s wort, try to avoid the sun as much as possible, or use sunscreen or wear protective clothing.63 Some of the other side effects linked to St. John’s wort include:64,65
As mentioned, St. John’s wort has the tendency to interact with multiple drugs, as highlighted by PennState’s Milton S. Hershey Medical Center:
If you’re taking these drugs, refrain from using St. John’s wort with them since it may lead to other health issues. This herb can also reduce the drugs’ effectiveness.66
Before using St. John’s wort, talk to a physician67 or mental health professional (if you’re struggling with a mental health problem) to determine if this will be beneficial for your condition and find out the ideal dosage you may need to take. If you’re pregnant or breastfeeding, avoid using any form of St. John’s wort,68 since it may cause your baby to develop drowsiness, fussiness or colic.69
Science has proven time after time that food is potent medicine. Broccoli, for example, has a solid scientific foundation showing it’s one of the most valuable health-promoting foods around. While it contains several health-promoting compounds, one of the most widely studied is sulforaphane.
The cancer-fighting properties of sulforaphane are perhaps the most well-known, but it has also been shown to benefit your heart and brain, boosting detoxification1 and helping prevent and/or treat high blood pressure,2 heart disease, Alzheimer’s3 and even autism.4,5,6 Now, researchers report sulforaphane may also be helpful in the treatment of schizophrenia.7,8,9
An initial study,10 published in Clinical Psychopharmacology and Neuroscience in 2015, involved just 10 outpatients with schizophrenia. Patients were given 30 milligrams (mg) of sulforaphane glucosinolate per day for eight weeks. As reported by the authors:
“Clinical symptoms using the Positive and Negative Syndrome Scale (PANSS) and cognitive function using the Japanese version of CogState battery were evaluated at the beginning of the study and at week 8.
A total of 7 patients completed the trial. The mean score in the Accuracy component of the One Card Learning Task increased significantly after the trial … This result suggests that SFN [sulforaphane] has the potential to improve cognitive function in patients with schizophrenia.”
More recently, a series of three animal and human studies11 by researchers at Johns Hopkins School of Medicine suggest sulforaphane may also benefit patients with schizophrenia by helping to rebalance the glutamate levels in their brain. As reported by Neuroscience News:12
“Schizophrenia is marked by hallucinations, delusions and disordered thinking, feeling, behavior, perception and speaking. Drugs used to treat schizophrenia don’t work completely for everyone, and they can cause a variety of undesirable side effects, including metabolic problems increasing cardiovascular risk, involuntary movements, restlessness, stiffness and ‘the shakes.’”
According to Dr. Akira Sawa, director of the Johns Hopkins The Schizophrenia Center, “It’s possible that future studies could show sulforaphane to be a safe supplement to give people at risk of developing schizophrenia as a way to prevent, delay or blunt the onset of symptoms.”13
One of the studies14 in this series, published January 9, 2019, in JAMA Psychiatry, assessed differences in brain metabolism between 81 schizophrenic patients and 91 healthy controls, finding schizophrenics had lower levels of key brain chemicals associated with the disease — glutamate, N-acetylaspartate,15 GABA and glutathione — in their anterior cingulate cortex, a brain region involved in executive function, emotional affect and cognition.16
According to the paper17 “Cognitive and Emotional Influences in Anterior Cingulate Cortex,” this brain region appears to be “the brain’s error detection and correction device,” and “is part of a circuit involved in a form of attention that serves to regulate both cognitive and emotional processing.”
In the brain, glutamate — an excitatory neurotransmitter18 — plays an important role in brain cell communication, and lower levels have been linked to both schizophrenia and depression.
Schizophrenics also had lower levels of N-acetylaspartate in the orbitofrontal region, an area involved in cognitive processing and decision-making, as well as the thalamus, an area involved in the relaying of sensory signals and the regulation of consciousness.
They also had lower levels of glutathione in the thalamus. Glutathione, a master antioxidant produced by your body, is made up of glutamate, cysteine and glycine, and is a physiologic reservoir of neuronal glutamate.19
For the second study in the series, the researchers focused on the management of glutamate in the brain. As reported by Neuroscience News,20 they wondered whether faulty glutamate management might be a key problem in the disease, and whether drugs could be used to “shift this balance to either release glutamate from storage when there isn’t enough, or send it into storage if there is too much.”
So, in this study,21 published February 12, 2019, in PNAS, they blocked an enzyme that turns glutamate into glutathione in the brain cells of rats, using a drug called L-Buthionine sulfoximine, thereby allowing glutamine to be used up.
“The researchers found that these nerves were more excited and fired faster, which means they were sending more messages to other brain cells. The researchers say shifting the balance this way is akin to shifting the brain cells to a pattern similar to one found in the brains of people with schizophrenia,” Neuroscience News 22 explains.
Next, to increase the level of glutamine stored as glutathione, they used sulforaphane, as it activates a gene that makes an enzyme required for the synthesis of glutathione from glutamate. As expected, this slowed the speed with which neurons fired.
In other words, it helped normalize the brain cells, allowing them to behave in a manner more like healthy controls. Dr. Thomas Sedlak, Ph.D., assistant professor of psychiatry and behavioral sciences told Neuroscience News:23
“We are thinking of glutathione as glutamate stored in a gas tank. If you have a bigger gas tank, you have more leeway on how far you can drive, but as soon as you take the gas out of the tank it’s burned up quickly. We can think of those with schizophrenia as having a smaller gas tank.”
In an earlier pilot study24 (counted as the third in this series) by the same team, published in the May 2018 issue of Molecular Neuropsychiatry, they used mice and healthy human subjects to assess the effect of sulforaphane on glutathione levels in the brain. Here, patients with a history of psychiatric illness were specifically excluded.
As explained by the authors:
“The participants completed two visits, scheduled 7 days (1 week) apart. The participants were given 100 µmol sulforaphane as standardized broccoli sprout extract in the form of 2 gel capsules, and instructed to ingest the extract each morning for 1 week …
Urine and blood specimens were collected prior to the first dose of broccoli sprout extract and within 4 h of the final dose. MRS [magnetic resonance spectroscopy] scans were performed prior to the first dose and within 4 h of ingesting the final dose …
Following 1-week administration of sulforaphane, the study participants demonstrated a significant augmentation of GSH in non-monocytes that include a mixture of T cells, B cells, and NK cells. The GSH level was 9.22 nmol/mL before sulforaphane administration and 12.2 nmol/mL following sulforaphane administration, a 32% increase …
We report that a short-term administration of sulforaphane was sufficient to significantly increase peripheral GSH levels in human subjects. We found an increase in GSH in the HP [hippocampus], but not elsewhere in the brain regions assessed. The peripheral GSH ratio had a strong and significantly positive correlation with brain GSH levels in the THAL [thalamus] upon sulforaphane treatment …
[I]n a submitted study, we will report that peripheral GSH levels may be correlated with cognitive functions. We thus posit the significance of exploring the possible correlations between peripheral GSH and clinical/neuropsychological measures and the influence of sulforaphane on such functional measures that are altered in neuropsychiatric disorders. The present study is a key first step toward such future studies.”
In summary, these findings suggest sulforaphane might be a safe alternative to help reduce psychosis and hallucinations in schizophrenic patients, although the researchers warn more studies are required to identify optimal dosing and assess long-term effects.
Another series of studies suggests cruciferous vegetables high in sulforaphane might benefit those with autism spectrum disorder (ASD), primarily by upregulating genes that protect against oxidative stress, inflammation and DNA damage, “all of which are prominent and possibly mechanistic characteristics of ASD,” the authors say.25
Sulforaphane also boosts antioxidant capacity, glutathione synthesis, mitochondrial function, oxidative phosphorylation and lipid peroxidation, while lowering neuroinflammmation. According to the researchers, these characteristics also make it suitable for the treatment of ASD.26
The first study,27 published in 2014, found daily treatment with dietary sulforaphane significantly reduced the severity of “socially impaired behavior” in children with ASD after 18 weeks. Improvements became obvious (compared to those in the placebo group) at four weeks of treatment.
At 18 weeks, the sulforaphane treatment group had a 34% reduction in Abberant Behavior Checklist (ABC) scores and a 17% reduction in Social Responsiveness Scale (SRS) scores. According to the authors:28
“[A] significantly greater number of participants receiving sulforaphane had improvement in social interaction, abnormal behavior, and verbal communication. Upon discontinuation of sulforaphane, total scores on all scales rose toward pretreatment levels.”
The second study,29 published in 2017, presented a case series follow-up of patients who continued the sulforaphane treatment after the first study ended. Here’s a limited outtake from the narrative provided by one of the families whose son is referred to as “R”:
“R’s parents wanted to help him: ‘He would make constant noises and did all these abnormal motor tics; [we] felt like he really had no control [over his behavior and body] and it was just noise, not functional words. He didn’t have any expressive language.’
R’s parents saw several medical specialists who prescribed a total of 18 different medications, all of which had either minimal or negative effects on R. ‘Nothing changed the constant noises or the terrible rage attacks,’ until R took SF [sulforaphane] …
R’s family took him to the Lurie Center at Massachusetts General Hospital where we were conducting the study on the effects of SF on males with ASD. The study was a randomized double-blind placebo-controlled trial. However, within days, R’s mother believed that he was taking SF:
‘I knew that he was on the study drug because I saw such a change so quickly. I want to scream from the rooftops and tell people to give the kids broccoli sprouts [extract] because literally, it changed my life,’ reported R’s mother.
‘Now we can go to the movies, restaurants, plays, we went on vacation with another family, we go to church, we just went to a concert, things we could never do before are now possible. [I am] able to have confidence and he [R] is more confident as well.’
N.B. Such a rapid response was unusual in the context of what was observed by the study physicians with other subjects. When responses to supplementation were observed, they generally took 3 or 4 weeks to become manifest. In this case, the study team actually wondered whether the mother might be exhibiting a placebo response; however, the ABC subscales and both ABC and SRS overall scores for R did also change.”
The third paper30 in this series, a trial progress report published in 2018, assessed the safety, clinical effects and mechanisms of action of sulforaphane in ASD. Interestingly, this paper describes how sulforaphane mimics “the fever effect” in ASD. This is where high fever temporarily improves behavior in autistic children. The researchers explain:
“Fever stimulates heat shock proteins (HSP) and cellular stress responses, leading to improved synaptic function and long-range connectivity. Expression of gene transcription by NFE2L2 (Nrf2), which is reduced in ASD, also increases during fever.
Sulforaphane (SF), an isothiocyanate obtained from broccoli sprouts, induces HSP and Nrf2 as well as ‘cell-protective’ responses that may benefit ASD through common cellular mechanisms underlying heterogeneous phenotypes.”
While this trial was still incomplete at publication, as only 46 participants out of a planned 50 had been enrolled, preliminary analysis showed “26% participants were much/very much improved at seven weeks, 38% at 15 weeks, 64% at 22 weeks, and 64% at 30 weeks,” the researchers said, adding that “preliminary results show that sulforaphane appears to be safe and effective in children with ASD.”
Sulforaphane may also be useful in the treatment of Alzheimer’s disease. In a 2018 study,31 mice with Alzheimer’s were treated with sulforaphane for four months, which significantly inhibited both the generation and accumulation of amyloid-beta, and alleviated several pathological changes associated with Alzheimer’s, including oxidative stress and neuroinflammation.
The mice also demonstrated cognitive benefits, remaining normal, cognitively speaking, compared to wild-type mice at 10 months of age, which is when dementia typically begins in Alzheimer’s mice. In tests of neurons themselves, pretreating cortical neurons with sulforaphane protected them against injury caused by amyloid beta.
An earlier study32 published in 2009 revealed that antioxidants — including sulforaphane — protect cells from oxidative damage, facilitate removal of the amyloid-beta peptide and reduce abnormal protein-related causes of disease.
In studying how sulforaphane interacts with amyloid-beta to prevent various neurodegenerative processes, researchers of a 2014 study33 used liquid chromatography/electrospray ionization mass spectrometry to reveal that amyloid-beta is less likely to aggregate in the presence of sulforaphane.
Another 2014 study34 showed that, in mice with Alzheimer’s-like lesions (induced in part by administration of aluminum), sulforaphane reduced neurobehavioral deficits by promoting the growth of new neurons (neurogenesis) as well as reducing the aluminum load.
While this article focuses on the neurological benefits of broccoli, research has revealed a long list of health benefits associated with this cruciferous vegetable, including a reduced risk for:35
Cancer — Studies have shown sulforaphane supports normal cell function and division while causing apoptosis (programmed cell death) in colon,37 prostate,38 breast39 and tobacco-induced lung cancer40 cells, and reducing the number of cancerous liver tumors in mice41
Insulin resistance45 and Type 2 diabetes46
Broccoli and other water- and nutrient-rich veggies also support healthy liver function, which in turn promotes optimal functioning of your natural detoxification systems. Broccoli sprouts, in particular, have been shown to help detox environmental pollutants such as benzene.50,51
This is important for virtually everyone these days, but especially women of childbearing age. Autistic children are known to have higher levels of environmental toxins in their system, and this underlying toxic burden plays a significant role.
Healthy liver function also helps promote healthy, beautiful skin, making broccoli a good antiaging food. What’s more, the sulforaphane in broccoli also helps repair skin damage.
To boost the benefits of sulforaphane in broccoli and other cruciferous veggies, pair them with a myrosinase-containing food.52 Myrosinase is an enzyme that converts the precursor gluocosinalate, glucoraphanin, to sulforaphane. Examples include mustard seed,53 daikon radishes, wasabi, arugula or coleslaw, with mustard seed being the most potent.
Adding a myrosinase-rich food is particularly important if you eat the broccoli raw, or use frozen broccoli. Ideally, broccoli should be steamed for three to four minutes to increase the available sulforaphane content. This light steaming eliminates epithiospecifier protein — a heat-sensitive sulfur-grabbing protein that inactivates sulforaphane — while retaining the myrosinase in the broccoli.54
Steaming is important, because without myrosinase, your body cannot absorb sulforaphane. If you opt for boiling, blanch the broccoli in boiling water for no more than 20 to 30 seconds, then immerse it in cold water to stop the cooking process.
If you prefer raw food, you’d be better off eating raw broccoli sprouts instead of mature broccoli. According to Dr. Paul Talalay, professor of pharmacology and co-author of the 1997 study55 “Broccoli Sprouts: An Exceptionally Rich Source of Inducers of Enzymes That Protect Against Chemical Carcinogens,” “Three-day-old broccoli sprouts consistently contain 20 to 50 times the amount of chemoprotective compounds found in mature broccoli heads.”56 As a result, you can eat far less of them while still maximizing your benefits.
More than 70% of U.S. water supplies have industrial-grade fluoride chemicals added under the guise of preventing tooth decay.1 The problem is that fluoride, a toxin, is linked to an increasing list of health damages, while the usefulness of ingesting it to prevent cavities is highly questionable.
Steven Gilbert, Ph.D., founder and director of the Institute of Neurotoxicology and Neurological Disorders (INND), works to bring awareness about the health effects of toxic substances, water fluoridation included.
In his “Connecting the Dots for Health” paper, he summarizes how connecting the dots between the science, history and ethics of water fluoridation clearly supports the action to discontinue water fluoridation in order to significantly reduce fluoride ingestion.2
If you’ve ever wondered how a neurotoxic chemical came to be added to U.S. water supplies, Gilbert states:3
“The history of community water fluoridation is a reflection of the post WWII era of the 1950’s when many thought chemicals in one form or another could solve almost any problem. Our gaze was focused on the beneficial properties of the chemicals, not on the potential hazards. A classic example is DDT, that in addition to being a potent pesticide, almost killed off predatory birds and more recently was found to be harmful to humans.”
In 1945, fluoride was given the green light by the U.S. government following the release of a large amount of hydrogen fluoride from DuPont’s Deepwater, New Jersey, plant. A massive quantity of toxic hydrogen fluoride was produced as a byproduct of industry, and its disposal was an inconvenient and costly problem.
To avert lawsuits, industry came up with the clever idea of revamping fluoride’s image — they told people fluoride was good for their teeth and began adding it to public water supplies. Initially, fluoride waste from the aluminum industry is what went into drinking water.
But by the late 1940s, they’d found a cheaper source — the phosphate industry, a byproduct of making fertilizer. According to a paper in Origins: Current Events in Historical Perspective, a production of The Ohio State University and Miami University departments of history:4
“Many are surprised to learn that unlike the pharmaceutical grade fluoride in their toothpaste, the fluoride in their water is an untreated industrial waste product, one that contains trace elements of arsenic and lead.
Without the phosphate industry’s effluent, water fluoridation would be prohibitively expensive. And without fluoridation, the phosphate industry would be stuck with an expensive waste disposal problem.”
Gilbert also explains that the decision to fluoridate U.S. drinking water was based on two studies comparing cavity rates in a city with fluoridated water (Grand Rapids/Muskegon, Michigan) with those in one without (Newburgh/Kingston, New York).
They were supposed to run for 10 years, but when some cavity reduction was seen in early reports, the U.S. Public Health Service approved water fluoridation after only five years — with no data on long-term toxicity.5
More than 300 studies have shown fluoride’s toxic effects on the brain,6 including a 2006 National Research Council review that suggested fluoride exposure may be associated with brain damage, endocrine system disruption and bone cancer.7
In 2012, Harvard researchers also revealed that children living in high-fluoride areas had significantly lower IQ scores than those who lived in low-fluoride areas8 and suggested high fluoride exposure may have an adverse effect on children’s neurodevelopment.
A study of Mexican women and children also raised concern, showing that higher exposure to fluoride while in utero is associated with lower scores on tests of cognitive function in childhood, both at the age of 4 and 6 to 12 years.9
Each 0.5 milligram per liter increase in pregnant women’s fluoride levels was associated with a reduction of 3.15 and 2.5 points on the children’s scores on the General Cognitive Index (GCI) of the McCarthy Scales of Children’s Abilities and the Wechsler Abbreviated Scale of Intelligence (WASI), respectively.
Fluorosilicic acid, which is the fluoride chemical added to drinking water, may also be contaminated with additional harmful compounds, including lead and arsenic. Children, in particular, are at risk from ingesting fluoride, but they are exposed to the same levels in drinking water as adults. According to Gilbert:10
“From the 1950s the PHS [Public Health Service] recommendation for the concentration of fluoridated water has been 1.0 mg/L (milligrams per liter or ppm) for most of the U.S., with a range of 0.7 to 1.2 mg/L. In 2015, this recommendation was lowered to 0.7 mg/L to reduce the toxic side effects of fluoride ingestion while attempting to maintain its beneficial effects.
For toxicological assessment, ingested doses are typically adjusted by body weight. Kids eat more, breathe more, and drink more than adults on a body weight basis so they will have higher fluoride doses than adults. Moreover, child organ systems such as the brain and bones are still developing, making them more vulnerable to the toxic effects of fluoride.”
In terms of overall toxicity, the Fluoride Action Network (FAN) describes acute fluoride exposure as more toxic than lead but slightly less toxic than arsenic.11 In fact, fluoride is a common ingredient in pesticides used to kill rodents and insects. Chronically, exposure to low levels of fluoride is also harmful, not only to your brain but to your body as a whole.
Fluoride is an endocrine-disrupting chemical, and studies have linked it to the rising prevalence of thyroid disease,12 which in turn can contribute to obesity, heart disease, depression and other health problems. Fluoride was once used to reduce thyroid function in people with hyperthyroidism (overactive thyroid), and even low doses of 2 to 5 mg may be enough to affect thyroid function.13
“This dose is well within the range (1.6 to 6.6 mg/day) of what individuals living in fluoridated communities are now estimated to receive on a regular basis,” FAN notes.14 A 2012 study also found a link between fluoride exposure and osteosarcoma, a rare type of bone cancer.15 A 2006 study also found a link between fluoride exposure in drinking water during childhood and the incidence of osteosarcoma among men.16
Such a link is biologically plausible, according to FAN, because bones are a principle site of fluoride accumulation, fluoride can be mutagenic at high enough concentrations and fluoride stimulates the proliferation of osteoblasts (bone-forming cells), which could increase the risk of malignancy.17
The majority of U.S. kids suffer from dental fluorosis, a discoloration and mottling of teeth caused by overexposure to fluoride in drinking water. While often brushed off as a cosmetic concern, this mottling is a sign of increased porosity of the enamel, and it’s permanent. If the tooth-forming cells are being harmed by fluoride, it’s likely that other cells in the body are too.
Research has found impairment in cognitive abilities among children with fluorosis (even mild fluorosis) compared to children with no fluorosis, for example.18 Studies have also found that children with higher levels of fluorosis have increased rates of cavities19 — a finding that suggests more is definitely not better, even when it comes to protecting against cavities.
According to Gilbert, “At a very mild or mild level, it causes white splotches or stripes on teeth. At moderate or severe levels, the mottling is more pronounced and can cause yellow or brown stains and pitting of the enamel, which can increase cavities.”20
According to the most recent data, the dental fluorosis rate in the U.S. is now a staggering 65 percent, with researchers stating, “The results of this study greatly increase the evidence base indicating that objectionable dental fluorosis has increased in the United States. Dental fluorosis is an undesirable side effect of too much fluoride ingestion during the early years of life.”21
Another study also revealed a more than 31% increase in the prevalence of dental fluorosis among 16- and 17-year-olds from 2011-2012 to 2001-2002. “The continued increase in fluorosis rates in the U.S. indicates that additional measures need to be implemented to reduce its prevalence,” those researchers concluded.22
The third piece of Gilbert’s puzzle is ethics, and from this perspective adding fluoride to U.S. drinking water is akin to drugging the majority of a population without its consent. Gilbert notes:23
“Physicians prescribe drugs on an individual’s needs, ensuring that it’s pharmaceutical grade (not contaminated) and requiring a specific dose for a specific length of time. They also must inform their patients of potential harmful side effects. However, the final decision on whether to take the drugs rests with the patient. With fluoridation, all these safety protocols are violated, taking away the individual’s right of informed consent.”
People who are more vulnerable to fluoride’s effects, such as infants, pregnant women or those with kidney disease and diabetes, have no way of avoiding this drug in their drinking water if they live in an area with fluoridated water.
While it’s possible to install a water filter, such as reverse osmosis, to remove fluoride from your drinking water, or obtain a separate source of drinking water, this puts low-income families, who may not be able to obtain these alternatives, at a disadvantage.
Considering there are many studies showing fluoride’s toxicity, the Precautionary Principle, which states that preventive measures should also be put in place to avoid exposure if there’s evidence of a substance causing harm, should be put into place.
“For these and other reasons, a growing number of public health professionals are recommending that fluoridation of drinking water be discontinued,” Gilbert says, supporting his recommended action to “discontinue water fluoridation so that ingestion of fluoride is greatly reduced.” This is the norm in most of the world, as about 95 percent of the world’s population drinks unfluoridated water.24
Finally, fluoride is not the answer to healthy teeth. A comprehensive oral care plan should include addressing your diet, reducing your net carb (total grams of carbohydrates minus your grams of fiber) intake and, if needed, taking nutritional supplements that support your oral health, such as vitamins C and K2, and coenzyme Q10.
Regular brushing with fluoride-free toothpaste and flossing are also important, as are regular professional cleanings with a mercury-free biological dentist.
By Anna Von Reitz
|(Natural News) In this study published in the journal Nutrition Research, researchers from Sungkyunkwan University in South Korea investigated the proliferative effect of apple ethanol extract on human adipose tissue-derived mesenchymal stem cells (ADSCs) and human cord blood-derived mesenchymal stem cells (CB-MSCs). They also identified the possible molecular mechanisms behind its beneficial effects. Tissue regeneration using…|
(Corey Goode) Dear Friends, I would like to publicly address recent events that have caused some concern within our community: namely, that I am allegedly the leader of a dangerous cult whose intent is to destroy people’s lives.