Case study reveals how cognitive decline can be reversed

Alzheimer’s disease, which is the most common form of dementia, eventually leads to the inability to carry out even the most basic of bodily functions, such as swallowing or walking. It is ultimately fatal, as conventional treatment options are few and universally ineffective.

Like autism among children, Alzheimer’s among seniors has reached epidemic proportions, with no slowdown in sight. On the contrary, evidence suggests the trend is worsening.

At present, Alzheimer’s affects an estimated 5.8 million Americans,1 and projections suggest the disease will affect 1 in 4 Americans within the next two decades. By 2050, Alzheimer’s diagnoses are projected to triple.2,3

And, while the U.S. Centers for Disease Control and Prevention lists the disease as the sixth leading cause of death in the U.S.,4,5 statistics published in the journal Neurology in 2014 revealed Alzheimer’s is vastly underreported on death certificates. In reality, the disease likely killed 503,400 American seniors in 2010,6 making it the third leading cause of death, right behind heart disease and cancer.7

The good news is that contrary to conventional claims, there are ways to prevent and even treat this tragic disease — not by drugs, but by diet and other lifestyle changes.

Dr. Dale Bredesen, professor of molecular and medical pharmacology at the University of California, Los Angeles School of Medicine, and author of “The End of Alzheimer’s: The First Program to Prevent and Reverse Cognitive Decline,” has identified a number of molecular mechanisms at work in Alzheimer’s, and created a novel program called ReCODE to treat and reverse it.8

100-patient case report sheds light on treatment options

Bredesen’s most recent publication is a case report9,10 of 100 patients using the ReCODE protocol. He has previously published three case reports, each involving just 10 patients. This fourth case report contains 100 patients treated at 15 different clinics across the U.S., all of which have documented pre- and post-cognitive testing.

Not only did all show improvement in symptoms, some of them also showed improvement in their quantitative electroencephalographs (EEGs). Others who underwent magnetic resonance imaging (MRI) with volumetrics also showed objective improvement.

“By all the criteria, these people showed improvement, subjective and objective,” Bredesen says. This is no small thing, as there is no conventional treatment that can reverse Alzheimer’s. There have been many drug trials to date, but all have failed to reverse the disease. As noted by Bredesen:

“There are a couple of medications, Aricept, Namenda … but these have a very, very modest impact. The most important thing is their improvement is not sustained. They don’t change the outcome of the disease. You get a little bump in improvement, then you go right back to declining.

The most important part of the [ReCODE] protocol … is that the improvement is sustained. You’re actually going after the root cause of what is causing the cognitive decline. That’s a big difference.”

Alzheimer’s is a protective response to inflammation

If one were to summarize Bredesen’s approach in one sentence, it would be “to improve the ratio between synaptoblastic and synaptoclastic activity, which is the brain’s ability to create new synapses versus destroying them.” In other words, the treatment allows your brain to create and maintain synapses again. Bredesen explains:

“The molecular biology of this disease shows that what we call Alzheimer’s disease is actually a protective response. It’s essentially a scorched-earth retreat.

You’re pulling back and saying, ‘We’re not going to let this insult kill us, so we’re going to scorch the earth so it (whether it’s bacteria or something else) cannot take advantage … of what’s there.’ You’re literally downsizing [your synapses]. As long as those insults are going on, you will be downsized.”

Beta-amyloid is a protein that is highly correlated with Alzheimer’s. However, all attempts at removing it have failed to improve the condition. Clearly, beta-amyloid in and of itself is not the primary cause, so simply getting rid of it is not the answer.

In Bredesen’s paper, he discusses the role of beta-amyloid as an antimicrobial peptide (AMP). Importantly, AMPs are critically important for host immunity. They target organisms such as bacteria, mycobacteria, viruses, fungi and protozoa. He explains:

“Here is the trick. It turns out amyloid beta is really part of the innate immune system. Its antimicrobial effect was first discovered and published by professor Robert Moir and professor Rudy Tanzi at Harvard.

This thing actually has, again, a protective response. Not only is it an AMP, but it also binds some toxins. For example, mercury, other divalent metals like iron and things like that. [Amyloid beta] has multiple effects. It is part of your response to insult.

When you take that into account, you realize it’s fine to remove amyloid, but please don’t do it before you remove all the insults. We’ve seen numerous people now who have had the amyloid reduced and gotten worse because the ongoing insults are still there.”

Most recently, the drug company Biogen halted its Phase II clinical trial for aducanumab, a drug designed to remove beta-amyloid, and this is the typical story for these kinds of drugs. And then a major trial of yet another approach to amyloid removal, the BACE inhibitor CNP520, was halted because the drug was associated with increased cognitive decline and brain atrophy.11

The protein refolding process is impaired in Alzheimer’s

About one-third of the proteins your body makes on any given day are misfolded. Thankfully, your body has a mechanism by which those misfolded proteins are refolded. Heat-shock proteins play a central role in this process, and if the misfolding is too severe, the heat-shock proteins help remove them altogether.

In fact, heat-shock proteins are a corollary of autophagy, the process by which your body cleans out damaged organelles. This relates to Alzheimer’s, because the refolding process is one of several factors that need to work in order for your brain to function. As noted by Bredesen:

“In all of these different neurodegenerative diseases, whether you’re talking about Alzheimer’s, Huntington’s, Lou Gehrig’s disease, Parkinson’s disease or Lewy body, they all feature proteins that are aggregated and that are typically misfolded. They are not degraded appropriately.

You lose not only the ability to fold but the ability to degrade these proteins. That is a critical piece. In fact, just recently, an article came out on a common neurodegenerative condition, newly described, which is called LATE, which is limbic-predominant, age-related TDP-43 encephalopathy.

In other words, this is a little bit like Alzheimer’s … [LATE] features TDP-43, which is a protein that is involved in numerous things, including protein folding … We lose that [protein-folding] ability as we start to downsize [synapses], as you don’t have an appropriate energy, you don’t have the appropriate trophic support.

You don’t have the appropriate hormonal and nutritional support … When we target ketosis, when we target insulin sensitivity, when we target mitochondrial support, that typically allows you to generate the appropriate ability to refold misfolded proteins …

You can induce the heat-shock response … by doing this combination of sauna and then [going] into the cold and then back to the sauna and then back to the cold …

You are recurrently activating this critical response [by doing that]. There’s no question it is going to be important, especially in ALS, but likely in all of the neurodegenerative conditions.”

The link between protein folding and cell death

As noted by Bredesen, there are three kinds of autophagy: macro-autophagy, micro-autophagy and chaperone-mediated autophagy. Each offers a slightly different way to repair, remove or recycle damaged organelles within the cell.

Specific proteins, for example, can be targeted for chaperone-mediated autophagy. Bredesen recounts findings of research he did to ascertain the linkage between protein folding and programmed cell death (apoptosis, where the entire cell is killed off and removed):

“If you fail to reform these [misfolded proteins], you literally activate an entire system that initially stops producing more protein. It’s basically saying, ‘We’re not keeping up with this. We’re going to shut this down.’ It attempts to refold. Then it attempts to destroy the proteins if it can’t refold them.

Then ultimately, if it cannot … keep up … it literally activates programmed cell death through specific caspases … This is something where you want to intervene upstream; understand why this is happening. And then if you’re unable to keep up with this, now, at least increase your heat-shock proteins so that you can refold. In this case, you prevent the induction of programmed cell death.”

Unfortunately, a vast majority of people do not have well-functioning autophagy, for the simple reason that they’re insulin-resistant. If you’re insulin-resistant, you cannot increase your adenosine 5’ monophosphate-activated protein kinase (AMPK) level, which prevents the inhibition of mammalian target of rapamycin (mTOR), and mTOR inhibition is one of the primary drivers of autophagy.

The case for cyclical fasting

While autophagy is clearly of critical importance, you don’t want to be in continuous autophagy. You also need to cycle through the rebuilding phase. One of the ways in which you can control this is through cyclical fasting. Bredesen typically recommends an intermittent fasting approach.

“You want to use appropriate fasting and an appropriate diet to activate this autophagy,” Bredesen says. “We recommend … 12 to 14 hours [of fasting] if you are apolipoprotein E4-negative (ApoE4-negative) … If you are ApoE4-positive, you’d want to go longer — 14 to 16 hours. There’s nothing wrong with doing a longer fast …

The reason we suggest longer for the ApoE4-positives [is because] if you are ApoE4-positive, you are better at absorbing fat. It tends to take longer to enter autophagy …

Typically, we recommend it about once a week. But again, a longer fast once a month is a good idea. It depends a lot on your body mass index (BMI). What we found is people who have higher BMIs respond better to this fasting early on. They’re able to generate the ketones.

If you lose both the carbohydrates and the ketones, you end up [feeling] completely out of energy … We are very careful when people are down below 20 on their BMI, especially the ones 18 or below. We want to be very careful to make sure to cycle them [in and out of ketosis] once or twice a week …

These are the ones where, often, exogenous ketones can be very helpful early on … Measure your ketones. It’s simple to do. We want to get you into, ultimately, the 1.5 to 4.0 millimolar [range for] betahydroxybutyrate. That is the goal.”

Test your ketones

So, to recap, while dementia patients with excess weight tend to respond favorably to cyclical fasting, at least initially, underweight patients may experience cognitive decline, as they’re simply too underweight to produce ketones in response to the fasting. For those who are underweight, Bredesen recommends using a ketone supplement such as medium-chain triglycerides (MCT) oil.

If that doesn’t bring you into the desired ketone level (1.5 to 4.0 mmol), or if it’s adversely affecting your low-density lipoprotein (LDL) particle number, he might recommend exogenous ketones — either ketone esters or salts. “We’d like to look at your LDL particle number and use that to titrate, to make sure that your LDL particle number is not too high,” he says.

To test your ketones, I recommend KetoCoachX.12 It’s one of the least expensive testing devices on the market right now. Another good one is KetoMojo. KetoCoach, however, is less expensive, the strips are individually packed and the device is about half as thick as KetoMojo’s, making it easier to travel with.

Energy demands are not met in neurodegenerative diseases

Nutritional ketosis, in which your body produces endogenous ketones (water-soluble fats), is important for all neurodegenerative diseases, but it’s not a complete cure-all. Bredesen explains:

“What we’ve come to realize from the research over the years is that neurodegenerative diseases, whether Alzheimer’s … macular degeneration … Lewy body, Parkinson’s or ALS, they all have one thing in common. They are related to specific subdomains of the nervous system.

Each of these has a unique requirement for nutrients, hormones, trophic factors, et cetera … In each case, there is a mismatch between the supply and the demand. For most of your life, you’re keeping up with that demand. With all of these diseases, you have a repeated or a chronic mismatch between the support and the requirement.

In Parkinson’s disease, it’s quite clear. You can create Parkinson’s disease simply by inhibiting mitochondrial Complex I. That specific subdomain of motor modulation, which is what Parkinson’s is all about, is the thing that is the most sensitive to reductions in mitochondrial Complex I support.

Therefore, when people have this, you need to bring the supply back in line with the demand. A critical way to do that is to supply the appropriate ketosis — the appropriate energy.

Now, if the person is continuing to be exposed to whatever chemicals are inhibiting Complex I — and it’s typically … mold-related biotoxins or organic toxins such as paraquat or glyphosate — as long as these are ongoing, you’re going to get very temporary relief.

The goal here is both to get rid of what is inhibiting Complex I and to flood the system, to help the system by giving appropriate support for the energetics … With Alzheimer’s, we’re really talking about a mismatch in trophic support. You’ve got this ongoing need as you’re making neuroplasticity.”

Why late-night eating is ill advised

Although I am not ApoE4-positive, I prefer fasting for 16 hours a day, essentially narrowing my eating window to just four to six hours. I also make sure to eat my last meal three to six hours before bedtime. One of the reasons for this advice is because avoiding late-night eating will increase your nicotinamide adenine dinucleotide (NAD+) levels, which are important for a variety of bodily functions.

Importantly, it will also reduce nicotinamide adenine dinucleotide phosphate (NADPH), which is essentially the true cellular battery of your cell and has the reductive potential to recharge your antioxidants. The largest consumer of NADPH is the creation of fatty acids.

If you’re eating close to bedtime, then you’re not going to be able to use the NADPH to burn those calories as energy. Instead, they must be stored some way. To store them, you have to create fat, so you’re basically radically lowering your NADPH levels when you eat late at night because they are being consumed to store your extra calories by creating fat.

Bredesen’s protocol includes this strategy as well. He calls his approach “KetoFlex 12/3,” because it generates mild ketosis and is flexible diet-wise. It can be done whether you’re a vegetarian or not. The 12/3 stands for a 12-hour minimum fast each day, and eating the last meal three hours before bedtime.

Certain supplements, including berberine, resveratrol, curcumin, quercetin and fisetin also boost autophagy, and can be used in addition to the nutritional timing. Bredesen explains:

“Sirtuin-1 (SIRT1) was identified as a critical molecule, both for longevity and has been studied extensively for its effects on longevity, but also for its effects on Alzheimer’s disease …

ApoE4 actually enters the nucleus and downregulates the production of this critical molecule, so you can see one of its many effects on Alzheimer’s disease. Well, when SIRT1 is made, it is actually made in an autoinhibitory fashion. It’s just like having a gun in a holster. It’s not active … NAD activates the SIRT1.

So does resveratrol. This is why people take resveratrol [or] nicotinamide riboside. These are both activating this program, which is moving you from … more of a pro-inflammatory approach to a longevity approach — a change in your metabolic pattern. That includes activating things like autophagy and also having an anti-Alzheimer’s and a pro-longevity effect …

[Q]uercetin also has an interesting impact on senescent cells … I think that that’s going to turn out to be an important way to impact a number of age-related conditions, including neurodegeneration.”

The drawback, and the reason you cannot rely on supplements alone, is that the bioabsorption of these polyphenols, like quercetin for example, is quite low. Oftentimes, you cannot absorb enough to get the full benefits.

Limit electromagnetic field exposures

There’s also convincing evidence showing electromagnetic field exposures (EMFs) such as that from cellphones and Wi-Fi play an important role. Bredesen agrees, and recommends his patients limit such exposures. In summary, EMFs activate your voltage-gated calcium channels, allowing the release of excess nitric oxide and superoxide in the cell, resulting in the creation of peroxynitrite.

Peroxynitrite causes similar damage to your DNA as ionizing radiation. It also damages your stem cells, mitochondria, proteins and cell membranes. Poly-ADP ribose polymerase helps repair DNA damage by extracting an adenosine diphosphate (ADP) molecule from NAD. Approximately 100 to 150 NAD are required to repair a single DNA break.

While this process works quite well, problems arise when continuous DNA damage requiring continuous PARP activation occurs, as this ends up decimating your NAD+ level. Bredesen adds:

“This is a critical area. The big problem we’ve had with this so far is that we can measure your NF-?B activation; we can measure your status of hormones, nutrients, magnesium, on and on and on. Typically, with our approach, we measure 150 different variables.

There is no simple way to measure the effect of EMF on a given person’s nervous system. I look forward to the day when we can do a test and say, ‘Aha. This person has 27.2 on their effects on their voltage-gated calcium channels because of EMFs.’ Because then we’ll really be able to alter that.

For now, the best we can say is — just as we go after biotoxins and chemotoxins — [EMF] is a physical toxin. The best we can say is, ‘Minimize that to the extent you can.’ You can certainly measure the exposure. We just don’t have a good way yet to measure its effect on your brain.”

More information

There’s no decline in sight for Alzheimer’s, at least in the foreseeable future, so it would behoove most people to just assume you’re headed for it and take action now, regardless of your age, to prevent it. When it comes to Alzheimer’s, prevention is surely far easier than trying to treat it once it has set in. As noted by Bredesen:

“This is all about prevention and early reversal. Those are the people where we see virtually 100% response. This is why I think there needs to be a global effort to decrease the burden of dementia. We’re just now starting a clinical trial. We’ve been trying to get institutional review board (IRB) approval for years …

It has finally been approved, so we’re starting a trial with Dr. Ann Hathaway, Dr. Deborah Gordon and Dr. Kat Toups, who are all seeing patients. We’re very excited to see what the trial will show with this approach. Because certainly, anecdotally, we’re hearing it all the time.

As you mentioned, we just published a paper a few months ago on 100 patients who showed documented improvement … I’m convinced we could, today, if everyone got an appropriate prevention, make this a very rare disease.”

Bredesen’s case report13 is open access, so you can download and read the full study. To learn more about Bredesen’s ReCODE protocol, see our previous interview, featured in “ReCODE: The reversal of cognitive decline.” In it, he reviews the various subtypes of Alzheimer’s, based on metabolic profiling, the influence of genetics, recommended screening tests and much more.

His book, “The End of Alzheimer’s: The First Program to Prevent and Reverse Cognitive Decline,” also provides the details, and would be a valuable reference in anyone’s health library.

You can also learn more about Bredesen and his work by following him on Facebook, Twitter or visit his website, Last but not least, keep an eye out for his latest book, “The First Survivors of Alzheimer’s.” This book, scheduled to come out toward the end of 2019, will feature first-person accounts from patients diagnosed with Alzheimer’s who beat the odds and improved.

An introduction to hepatitis C

Discovered in 1989,1 hepatitis C is a dangerous liver infection caused by the hepatitis C virus, which can spread through blood-to-blood contact, such as through blood infusions or sharing of contaminated needles. It ranges in severity — the disease can manifest as a mild illness that lasts only a few weeks or months, or it may progress into a serious lifelong illness.2 Discover more important facts about this disease.

Hepatitis C attacks your liver

The liver is one of the hardest-working organs in your body, as it performs a variety of functions. According to WebMD, it works with other organs to digest food and secretes bile, although its main role is to filter out and eliminate harmful toxins from your blood.3 It’s even referred to as the “Grand Central Station” of your body — all the nutrients that enter your intestines go through the liver.4

So, if a potentially dangerous disease like hepatitis C threatens to destroy your liver — and your overall health — then you need to take the necessary actions to ensure that your liver continues to function properly.

Hepatitis C statistics: How prevalent is this disease?

Approximately 2.4 million people in the country are dealing with chronic hepatitis C,5 although a 2015 study notes that the number could be higher — as much as 4.6 million.6 Of all these infected, 51% are unaware of their condition — until it’s too late.7

The numbers are more alarming globally. In 2015, an estimated 71.1 million people worldwide are dealing with this disease.8 Hepatitis C cases are highly prevalent in areas like North Africa, Central and East Asia and the Middle East.9 According to a 2016 World of Gastroenterology study, the countries with high rates of chronic hepatitis C infections include Egypt (14.7%), Iraq (3.2%) and Yemen (2.2%).10

Hepatitis C patients show no symptoms until the disease has already worsened

It can be difficult to diagnose acute hepatitis C because it lacks definitive symptoms. In fact, 60% to 70% of people who become infected are asymptomatic, or exhibit no symptoms.11 The incubation period (time from exposure to the onset of symptoms) is between two weeks and six months.12

Should symptoms manifest, they are usually similar to those of other viral infections. These include abdominal pain, joint pain, fever, nausea and fatigue. In some cases, jaundice (yellowing of the skin and eyes) and clay-colored stools may also be seen.13,14

However, once the virus stays in your blood for a year after the acute infection period, it becomes a chronic infection. This is what makes this disease extremely damaging because if left untreated, it can lead to cirrhosis, liver failure or liver cancer.15 Hepatitis C is one of the most common causes of chronic liver disease.16

If you love your liver and your health, read these articles right away

Because there are usually no telltale signs until it’s too late, hepatitis C is a potentially dangerous disease that you should be aware of. Staying well-educated about this ailment, plus learning to listen to your body for potential warning signs, is the best way to avoid hepatitis C or, if you already have it, severe complications from it.

These articles will provide you with substantial information about hepatitis C and help you learn more about its symptoms, risk factors, causes and what hepatitis C can do to your body. You’ll also discover useful pointers on how you can live with hepatitis C, as well as how to prevent this ailment from damaging your liver — and overall health.


Hepatitis C: Introduction

What Is Hepatitis C?

Hepatitis C in Pregnancy

How Do You Get Hepatitis C?

Hepatitis C Duration

Is Hepatitis C Contagious?

Hepatitis C Causes

Hepatitis C Types

Hepatitis C Symptoms

Hepatitis C Treatment

Hepatitis C Prevention

Hepatitis C Diet

Hepatitis C FAQ

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What Is Hepatitis C?

How to Cook Lobster Tail

When people think of lobster, they usually associate it with extravagance. According to Business Insider, restaurants typically charge $30 to $45 for a single lobster. These prices can turn many people away from lobster, which is delicious and contains astaxanthin, a carotenoid with various health benefits.

The expensive prices are attributed to the simple fact the crustaceans are harvested from the wild because there are no commercial lobster farms. Fishermen have to wait for lobsters to mature, which takes about seven years before they are legal harvesting size. In addition, once gathered, lobsters must be kept alive and fresh — thus adding to shipping costs — because they are susceptible to bacterial disease, which can spoil the meat quickly if the lobster dies.

How to Prepare Lobster Tail

If you’ve decided to purchase lobster to cook at home, the tail is a good place to start. The process starts with choosing the best meat first. According to BBC Food, there are two main species of lobster used for cooking: the American (or Maine) or European lobster. Maine lobster is considered the largest and the best, while the European lobster is smaller. However, both can be used in recipes depending on what you can find.

Buying lobster tails requires discerning eyes so you can get the most out of your money — after all, lobster is an expensive food. When looking for lobster tails, first make sure they come from reputable harvesters, then follow these additional tips:

  • Cold water lobster tails are commonly sold in grocery stores and restaurants in North America. They are generally more expensive because of the higher-quality meat compared to warm water lobster tails.
  • An alternative to American lobster tails are warm water lobster tails, usually from the spiny Caribbean or rock species.
  • To manage your budget, keep an eye on the weight of the lobster tail. The heavier the package, the more expensive it becomes.
  • Avoid lobster tails that look yellow or gray. Avoid meats with glazing as well (a process where water is injected to the tail to increase shelf life during shipping).

Once you’ve purchased lobster tails you’re satisfied with, here’s how to prepare the meat to get the most out of your purchase, according to Better Homes & Gardens:

  • In one hand, hold the lobster tail. With the other hand, cut lengthwise through the center of the shell and the meat using kitchen shears. Stop cutting as you reach the end of the shell, which is the wide end of the tail.
  • Spread the cut further apart, then loosen the meat with your fingers.
  • Separate the meat using your thumbs, but keep some of it attached near the end of the tail.

How Long Does It Take to Cook Fresh Lobster Tail?

The cooking time for fresh lobster tail depends on the recipe, the size of the lobster tail and your chosen cooking method. Working Mother offers the following approximates for cooking lobster tails:

  • Boiling — Lobster tails are generally boiled for three minutes and 30 seconds to four minutes.
  • Steaming — Lobster tails usually cook for eight to 10 minutes when steamed.
  • Broiling — It usually takes 10 to 15 minutes to broil lobster tails.
  • Grilling — Grill the lobster tail flesh side down for two to three minutes, then flip with the shell side down and cook for another six to eight minutes, transferring it to a cooler part of the grill.

How Long Does It Take to Cook Frozen Lobster Tail?


Better Homes & Gardens recommends placing your lobster tail in the refrigerator overnight for eight to 10 hours. If you’re in a hurry or forgot to thaw your lobster tails overnight, Working Mother indicates that you can quickly thaw your tails in a bowl of water and defrost them in an hour or so.

Ideally, lobster tails should be thawed because it results in more tender meat once cooked. However, you can still cook your lobster tails frozen, although you will need to add a few minutes to your cooking time. Larger tails may need double the cooking time, if cooked frozen.

Various Ways to Cook Lobster Tails

Lobster tails are not well-known just because they’re expensive — many people like them for their flavor and the myriad ways they can be prepared. If you haven’t cooked lobster tail before, the following guides can help you get started.

Grilled Lobster Tail

Lobster tails can be grilled, but they require certain preparations to cook them appropriately. Here’s a recipe adapted from the book “Simply Grilling: 105 Recipes for Quick and Casual Grilling” to help you enjoy grilled lobster tails in the best way possible:
Grilled Lobster Tails With Drawn Butter


  • To make the drawn butter, melt it in a small saucepan over medium-low heat. Cook until the solids have separated. Discard the milk solids, leaving behind the drawn butter.
  • Heat the grill to medium-high. Brush the grill lightly with coconut oil.
  • Boil the lobster tails in high heat for six minutes.
  • Butterfly each tail by cutting on the underside of the shell without damaging it. Spread the halves to expose the meat, then brush the flesh with oil. Sprinkle seasoning on top.
  • Grill the lobster with the flesh facing down. Cook for three to four minutes per side. Remember to close the grill after flipping.
  • Serve with the drawn butter on the side.

Baked Lobster Tail

If you’re looking to explore a new way of cooking lobster tails, baking is the answer for you. Here’s an overview on how to prepare lobster tails for baking:

  • Thaw lobster tails and heat oven to 425 degrees Fahrenheit. Pat the tails dry with paper towels and prepare the tails by butterflying them.
  • Sear the lobster tails first by grilling them in a hot pan for three to four minutes, then transfer them to the oven in a baking pan, with the meat facing upward.
  • Bake the lobster tails for 10 to 12 minutes or until the meat is opaque.

Boiled Lobster Tail

Boiling is one of the most common ways of cooking lobster tails, as it’s an easy way to prepare them. Here’s an overview on how to boil lobster tails according to Better Homes & Gardens:

  • Thaw the lobster tails first, then dry with paper towels. Prepare the shells by cutting and spreading them apart.
  • Boil 6 cups of filtered water (with Himalayan salt) in a 3-quart saucepan. This amount is good for four 8-ounce lobster tails.
  • Simmer the lobster tails for eight to 12 minutes, without the cover, or until the shells become bright red and the meat is tender. Drain.

Steamed Lobster Tail

Steaming lobster tails is quite similar to boiling them. The difference is that you don’t need to watch over the water you’re boiling. To try this method, simply follow these steps:

  • Insert a steamer rack into a large pot for your lobster tails.
  • Bring water to a boil, then place the lobster tails inside.
  • Cover the pot and cook for eight to 10 minutes.

Fried Lobster Tail

Lobster tails aren’t usually fried, but it’s doable. By frying, you can dip the tails in breading, creating a crispy food reminiscent of fried fish. Here’s a simple recipe adapted from Just a Pinch Recipes:

  • Lobster tails (the amount is up to you)
  • For the egg wash, 1 egg and 1 cup of buttermilk
  • Coconut oil for frying

Tempura batter:

  • 1 cup arrowroot flour
  • 1 tablespoon arrowroot starch
  • 1 and 1/2 cups water
  • 1/2 teaspoon Himalayan salt
  • 1/2 teaspoon organic cayenne pepper
  • 1 teaspoon organic garlic powder

Clarified butter:

  • 2 sticks raw grass fed butter


  • Beat the eggs and buttermilk. Set aside.
  • Mix all tempura batter ingredients. Set aside.
  • Cut the lobster tail meat into bite-sized chunks, then dip them into the egg wash. Afterward, dip them into the tempura batter.
  • Fry each piece for three to four minutes in hot coconut oil.
  • Remove from the pan and transfer to a paper plate.
  • Melt the butter in a separate saucepan under low heat. Let stand for five minutes, skimming the foam on top into a dipping container. Discard the milky solids.

Lobster Tail Recipe: Garlic Lobster Tails

Aside from the other recipes mentioned above, here’s a keto-friendly lobster recipe you can try, adapted from Top Notch Nutrition. It has few ingredients and very little cooking time, making it perfect for beginners:


  • Two 4- to 6-ounce lobster tails, fresh
  • 2 tablespoons coconut oil
  • 2 tablespoons garlic, minced
  • 1 teaspoon garlic powder
  • Paprika to taste
  • 2 tablespoons melted raw grass fed butter (for dipping after cooking)


  • Heat your broiler on low.
  • Cut the shell of the tail down the center.
  • Open the shell to lift up the meat. Once it loosens, place the meat on top of the shell and close the shell under the meat.
  • Drizzle the lobster meat with coconut oil. Sprinkle on the garlic powder and paprika.
  • Put the shells in a baking pan and place the minced garlic under the tails.
  • Broil the tails for one minute per ounce.
  • Dip the lobster tails in melted butter while eating.

Enjoy Lobster in Many Ways

Lobster tails are one of the most diverse foods you can cook. Learning how to cook lobster tails will help you expand your culinary knowledge and give you a delicious meal to enjoy.

Frequently Asked Questions About Cooking Lobster Tail

Q: How can you tell when lobster tail is done?
It’s always important to cook lobster properly, as undercooking can expose you to foodborne pathogens. The best way to tell if lobster tails are cooked properly is if the flesh is opaque.

Q: Should you thaw lobster tails before cooking?
Thawing lobster tails results in more tender meat once cooked.

Q: How do you defrost lobster tails?
Lobster tails should be thawed in the refrigerator overnight.

Q: Do you cut the lobster tail before you steam it?
Yes, you can butterfly lobster tail before cooking to help cook the meat better. This is explained in the different recipes listed in this article.

Why I Oppose Manna World Holding Trust, Etc.

By Anna Von Reitz

In 25 words or less:  the culprits responsible for this situation — all of whom are thieves guilty of capital crimes — chose one of their own to be the Holder, instead of returning control of the assets to the actual owners. 

Is that simple enough for everyone to understand?  

Kim Goguen is one of them by blood, by training, by indoctrination, by belief, by all that is endemic to the evil that pervades the Banking Cult.  

She may try to do things differently.  She may have good intentions.  But that does not change the fact that she is in control of (or claims to be in control of) stolen goods and assets that don’t belong to her, and she is standing there bare-faced and refusing to give the assets back to the actual owners. 

So, what am I as a Justice supposed to do?  Support her in unlawful possession?  Just because she seems to be a Nice Person?

Looking deeper into the situation, it is clear that the actual Holder of all these assets is and should be The United States of America, which means that they fall under my Fiduciary Office and not hers.  She should be working for me and doing what I tell her to do, not stomping around making False Claims based on possession related to piracy, fraud, and unlawful conversion.   

What I would tell her to do is very simple — start releasing assets in a step-by-step fashion to the people they actually belong to.  

Return all the Titles to American Land to the most recent American Owners, meaning living people, not corporations.  Do the same on a worldwide basis.  Return all the “individual” ESTATES to the Americans they are named after, all the Brits they are named after, all the Germans they are named after…. .  

It isn’t rocket science.  The benefit of the massive wealth stolen and amassed by these criminals can be returned to the people individually and as funding for many beneficial technologies and new infrastructure.  

And as for the military, I’d tell them to stop goosing around like idiots. Tell the Europeans we are calling in their debts and taking over their corporations and their banks if they don’t shut down the B.E.A.S.T. system and the UN nonsense and stop all the False Claims against Americans.  

You military generals may be “US Citizens” all you like, but you need us to back you up and give you a contract, otherwise you are all just a bunch of mercenaries and thugs with no standing as soldiers and if you are going to betray the people who actually employ you and do the bidding of Middlemen acting in Breach of Trust —- then why do we need you?  Hello?  Why do we need to keep paying you and buying you new toys?  

What good are your oaths to protect and defend “the” Constitution when you are too clueless to know which Constitution that is?   Too clueless to know that you work for the American People, not the Queen, not the Pope and not the ever-loving “UN CORP”, either? 

NATO is the Enemy. It was formed by the same group of criminals that formed the European Common Market and that are trying to create the B.E.A.S.T system.  

The US Navy is compromised and in conflict of interest.  Even the Marine Corps that actually allows that our troops are American  — is compromised in its command structure because they take their orders from the Navy and the Navy takes their orders from the Queen.   We have no Coast Guard thanks to the same cozy arrangement.  

The Coast Guard is supposed to be protecting us –Americans– from Inland Piracy.  That is what we pay them to do as their principal responsibility.  But guess what?  All this graft and fraud has come ashore on their watch.   They are stood down, useless, helpless to stop the human trafficking and gun running and drug running, left without funding just like the Federal Marshals who are supposed to be protecting us and our international land jurisdiction.  We have to rely on volunteers to provide enforcement. 

They– the Navy, the Marines, and the Coast Guard (inappropriately)  are all under the command of the Brits, and the Queen is guilty and up to her knickers in graft and fraud against her own people and the “former” commonwealth nations and us, too, taking the payola and not giving a fig more than her batty great-grandmother did about the consequences of her betrayals.  

The pelicans at the BBC snarking about Trump and trying to get something started with Iran — or anywhere else they can get a nice little war going — are nothing but a bunch of greedy self-interested war mongers and propagandists for men who make their money on war.  

So let’s throw them in front of the cannons and the tanks, this time, shall we?

Haul out all the BBC Commentators and the Board Members of all of Britain’s top corporations and their US Brethren and the members of Congress, and let them fill the trenches. 

They always want war, they are always pulling false flags and blaming others for their misdeeds. It’s time that the Brits either shut up and pay up, or we stand back and let the buggers take it square in the shorts and nobody — not us, not the Aussies, not the Canadians, not the New Zealanders, not the Scots, and certainly not the Irish — nobody comes to rescue them. 

Anyway that “Special Relationship” with the British  is why Trump has had to fall back on the US Army and Joint Forces Command of the American Armed Forces.  See that word?  American?  They aren’t talking about South Americans, in case you were wondering. 

Trump can’t trust the US Navy.  Its a sad day when the Commander-in-Chief of American Armed Forces can’t depend on a third of the military we pay for, but that is the circumstance. The Navy wags have tired to kill him three times that I know of, and also tried to get WWIII started with the Russian submarine incident.  I can only thank God that Putin had sense enough to see through it and Trump had sense enough to be skeptical and not rush into anything.  

All you Brass in the Navy?  You’d better straighten your gigs and ratlines. The Queen is supposed to be acting in Good Faith under our Delegated Authority, but instead, she’s in Breach of Trust.  Be aware.  She’s not your actual Boss. We are.  Without us, all she can do is serve you tea and crumpets and shower you with “knighthoods” like she gave the feckless traitors Colin Powell and  G.H.W. Bush and Henry “the Floss” Kissinger. 


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And We Know VIDEO 7-27-19… “SerialBrain2: The secret message behind Ivanka Trump tweeting ‘United Kingston’”

[Kp update: some might have an interest in going to 22 minutes of the video to learn more about what has been going in to the Morgellons chemtrails. According to the presenter, these are actually beings that are “sucking” energy. (full video link)]

As before, I’m finding it much easier to understand the SB2 articles by viewing these AWK videos. Another wonderful job by And We Know. This video is based on the SB2 article, “The secret message behind Ivanka Trump tweeting “United Kingston”” (related Kp blog post).

[Kp note: although these decodes can be very, very deep, I feel each of them can encourage learning to use one’s own discernment and find what “rings the Inner Bell”. Personally, I find it useful to both read the SB2 posts and view the And We Know videos (Bitchute channel)… I find I get more complete data input that way.]
Crazy Bernie:
The Chemtrails, Morgellons, Smart Dust and Archons Connection:
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U.S. Congress Votes for Pentagon to be Investigated for Using Insects as a Biological Weapon

by Aaron Kesel

In a real-life X-Files episode, United States House members are reportedly concerned in a bipartisan vote that the Pentagon may have unleashed biological weapons or entomological warfare in the form of ticks or other insects that caused the spread of Lyme disease.

Roll Call, a congressional-focused newspaper reports that on July 11th the House secretly decided through a voice vote to support adding an amendment to the 2020 defense authorization bill that would require the Department of Justice to look into whether the Pentagon/ CIA/ DoD umbrella weaponized ticks.

According to Roll Call, New Jersey Republican Rep. Christopher Smith wrote the amendment demanding the inspector general “conduct a review of whether the Department of Defense experimented with ticks and other insects regarding use as a biological weapon between the years of 1950 and 1975.”

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