By Anna Von Reitz
By Anna Von Reitz
By Anna Von Reitz
There are two prevailing theories about the origins of SARS-CoV-2. The first is that SARS-CoV-2 emerged naturally and jumped from wildlife to humans, with or without an intermediary host. The other is that the virus was being kept and/or studied in a lab, from which it escaped.
Peter Daszak, president of EcoHealth Alliance, is one of the most ardent supporters of the natural origins theory. He told The Associated Press in November 2020 that SARS-CoV-2 could have passed from a wildlife poacher to a trader who brought it to Wuhan.1 Daszak also organized the publication of a scientific statement, published in The Lancet and signed by 26 additional scientists, condemning such inquiries as “conspiracy theory.”2
Daszak, however, is part of the World Health Organization team that investigated the origins of SARS-CoV-2 and has a long history of close ties to the Chinese laboratory in question — the Wuhan Institute of Virology (WIV), from where it appears increasingly likely that the virus emerged.
The video above, “Deception in America Episode One: The Tale of Peter Daszak,” presents a succinct history of his involvement, as well as that of Dr. Anthony Fauci, director of the National Institute of Allergy and Infectious Diseases (NIAID), which is part of the National Institutes of Health (NIH) and who has also long backed dangerous coronavirus research, including that conducted by EcoHealth Alliance. According to the film:3
“From the beginning of the COVID-19 outbreak, said to have begun some time in December of 2019, the battle to control the narrative has been fought ferociously by global organizations, CEOs, billionaires and the programs at the center of the Wuhan Institute of Virology controversy … Despite his attempts to shift the blame to anyone else but himself and his team, all roads eventually lead back to Daszak.”
EcoHealth Alliance is a nonprofit organization that receives millions in funding every year from NIAID, and then gives it to WIV. In fact, the film reveals that, since 2002, EcoHealth Alliance and Peter Daszak have received over 32 grants worth hundreds of thousands of dollars from NIH, with most coming from NIAID:4
“Over the years, Peter Daszak and the Ecohealth Alliance have received $5,764,128 from the Fogarty International Center and $7,875,012 from the NIAID, for a grand total of $13,639,140.”
EcoHealth Alliance also has contracts with other government departments worth millions of dollars. The following contracts are among them:5
There are many others as well. According to USASpending.gov, EcoHealth Alliance has 36 contracts with various government agencies.6 Over the years, the organization has been awarded $61.5 million, with DOD chief among its funders, giving them a total of $41.9 million. Overall, 91% of EcoHealth Alliance’s funding comes from government grants.7
Daszak has extensive connections, including having worked for the Center of Infection and Immunity at Mailman School of Public Health at Columbia University, alongside colleague Jeffrey Sachs, the former director of The Earth Institute at Columbia.8 Sachs, also an adviser to the United Nations, spearheaded the Millennium Villages project, which was an attempt to reduce extreme poverty in Africa, supporting a shift to self-sufficiency.
While the project claimed to be a success, an evaluation in 2012 revealed its goals were unrealistic and serious questions were raised about the project’s ability to create long-lasting impact, along with its cost-effectiveness.9
Sachs also wrote a number of articles in support of China, stating, for instance, that “the U.S., not China, is the real threat to international law,”10 and was named chair of the Lancet Commission on COVID-19. He then named Daszak as one of the commissioners.
“Peter Daszak’s constant involvement with people like Jeffrey Sachs and the World Economic Forum suggests that he could be another one of those types of men who are so disconnected with reality that they will do anything to shape the world, not realizing what they are doing,” the film noted.11
In fact, in a Tweet from May 10, 2011, EcoHealth Alliance wrote, “Q for the crowd: is western style democracy, a la USA, compatible with sustainability? Or is eastern style (a la China) better?”12 As further noted by the film:13
“Peter Daszak’s Ecohealth Alliance receives far more in funding from the U.S. government, including grants from the Department of Defense, among others. He is also closely associated with Jeffrey Sachs via Columbia, who frequently appears alongside George Soros in talks and seminars.
Daszak is very well connected to many globalist enterprises and their organizers. His own organization touts the sustainable development goal model, originally devised by Jeffrey Sachs.
So, when Peter Daszak deflects and says his team and their research that are directly involved the Wuhan Virology Lab has nothing to do with the SARS coronavirus outbreak in 2019, it follows a pattern that the organizations he surrounds himself with do, wherein they blame a lack of funding, or other outside factors. It is never his fault.”
Gain-of-function (GOF) research refers to studies that have the potential to enhance the ability of pathogens to cause disease, including enhancing either their pathogenicity or transmissibility.14 Such research is by its very nature controversial, since there are clear risks should the information be misused or the pathogens escape (or are maliciously released).
Jonathan Latham, Ph.D., a molecular biologist and virologist and Allison Wilson, Ph.D., a geneticist, are among those who believe gain-of-function research performed at WIV played “an essential causative role in the pandemic.”15 However, Daszak continues to say emerging infectious diseases come from climate change and ecological drivers, ignoring the gain-of-function research that he and his organization are directly involved with.
For instance, as reported by Alexis Baden-Mayer, political director for the Organic Consumers Association, EcoHealth Alliance lists WIV and the Wuhan University School of Public Health as subcontractors under a $3.7-million NIH grant16 titled, “Understanding the Risk of Bat Coronavirus Emergence.”
EcoHealth Alliance also used a sub-grant17 from the University of California at Davis to fund a gain-of-function experiment by Shi Zhengli, Ph.D., the director of WIV’s Center for Emerging Infectious Diseases, also known as “bat woman,” and colleague Ralph Baric from the University of North Carolina at Chapel Hill, involving the use of genetic engineering to create a “new bat SARS-like virus … that can jump directly from its bat hosts to humans.” According to Baden-Mayer (see hyperlinked article above):
“The work, ‘A SARS-like cluster of circulating bat coronaviruses shows potential for human emergence,’18 published in Nature in 2015 during the NIH’s moratorium19 on gain-of-function research, was grandfathered in because it was initiated before the moratorium … and because the request by Shi and Baric to continue their research during the moratorium was approved by the NIH.
As a condition of publication, Nature, like most scientific journals, requires20 authors to submit new DNA and RNA sequences to GenBank, the U.S. National Center for Biotechnology Information Database. Yet the new SARS-like virus Shi and Baric created wasn’t deposited21 in GenBank until May 2020.”
Daszak and WHO officials continue to state that safety guidelines make it very unlikely that SARS-CoV-2 could have escaped from a lab, but a paper published in the Bulletin of the Atomic Scientists22 revealed in 2012 that it’s a matter of when, not if, a potential pandemic pathogen (PPP) escapes.
At the time, they noted that there were at least 42 facilities engaged in research on live PPPs, “and the actual number is likely higher.” Using a conservative estimate that the probability for escape from a lab in a year is 0.3%, they found:
“[This] translates to an 80 percent likelihood of escape from at least one of the 42 labs every 12.8 years, a time interval smaller than those that have separated influenza pandemics in the 20th century. This level of risk is clearly unacceptable.”
In fact, biosecurity breaches in high containment biological labs in the U.S. and around the world have occurred with surprising frequency,23 and as the film noted:24
“It is hardly reassuring that despite increased policy demands for rigorous biosecurity procedures, potentially high-consequence breaches occur nearly daily. In 2010, 244 unintended releases of bioweapon candidate ‘select agents’ were reported. Being practical, the question is not if such escapes will result in a major civilian outbreak, but what the pathogen will be and how well it can be contained, if it can be contained at all.”
WHO’s investigative commission, tasked with identifying the origin of SARS-CoV-2, announced the Wuhan Institute of Virology and two other biosafety level 4 laboratories in Wuhan, China, had nothing to do with the COVID-19 outbreak in February 2021. Since then, WIV deleted mentions of its collaboration with the NIAID/NIH and other U.S. research partners from its website.25 It also deleted descriptions of gain-of-function research on the SARS virus.
Several members of the U.S. Congress have now vowed to launch their own investigation to explore the lab accident theory. The Energy and Commerce Committee has also requested extensive records from both the NIH and EcoHealth Alliance detailing research and collaborations with WIV.26
If SARS-CoV-2 did, in fact, come from a lab, it shows clearly that gain-of-function research is the real threat and reason for locking down the healthy and highlights the disturbing truth that any such pathogen manufactured to infect humans can be designated as a biological weapon, even if it was created with non-nefarious intentions.
With each passing year, more people are affected by seasonal allergies. In the quest to identify the mechanism behind the growing number of people experiencing allergies, scientists have identified several factors that play an important role. One of these is air pollution.1,2
According to the Asthma and Allergy Foundation of America,3 more than 50 million Americans have experienced a variety of types of allergies each year, and that number only continues to rise. The annual cost exceeds $18 billion for allergic rhinitis and $25 billion for food allergies.
The most common types of seasonal allergies are reactions to tree, grass and weed pollen. Allergic rhinitis, which is also called hay fever, affects 5.2 million children and 19.2 million adults. Although these numbers are staggering, scientists expect them to continue to rise as increasing levels of carbon dioxide (CO2) have affected plant growth and pollen production.
The most common symptoms of allergic rhinitis include sneezing, stuffy, runny nose, watery and itchy eyes and itching in your nose, throat or mouth. A sizable number of allergy sufferers experience noticeable brain fog as well. Food and seasonal allergies are your body’s reactions to particles it considers foreign.
The first time your body meets a protein allergen, plasma cells release immunoglobulin E (IgE).4 This attaches to the surface of mast cells, which are found in great numbers in your surface tissue such as skin and nasal mucus membranes. Mast cells release important chemical mediators, one of which is histamine.
The next time your body encounters this allergen, your mast cells become activated within minutes and release a powerful cocktail of histamines, leukotrienes and prostaglandins. This triggers the cascade of symptoms that you associate with allergies.
One of the major components of air pollution is carbon dioxide. According to the Center for Climate and Energy Solutions,5 carbon dioxide makes up 76% of all greenhouse gas emissions. Trees and other plants use carbon dioxide for photosynthesis. You might think rising levels of carbon dioxide may be good for plant life, but like most things, plants need a balance.
Gilles Oliver is an engineer from the National Aerobiological Surveillance Network, which tracks pollen throughout France. He spoke with a reporter from Vice France about the interaction between CO2 and plant life, saying,6 “But when the proportion of this CO2 in the air increases, plants grow faster and produce more pollen.”
An increase in pollen production raises your exposure to pollen, and thus produces more severe seasonal allergies. Changes in temperature and CO2 have also extended the frost-free season and thus lengthened pollen season.7 However, the pollen being produced by these plants may be contributing to the collapse of the bee population.
Scientists have found that increases in CO2 produce more pollen, but the pollen has lower protein levels.8 When bees harvest from flowers they first drink the nectar and then collect the pollen. Nectar is high in carbohydrates, while pollen offers long-term nutrition and provides the bees’ only natural source of protein.
When scientists compared flowers from Canada Goldenrod collected from 1842 to the present, they found the percentage of protein dropped by 33%.9 Over the same number of years, CO2 levels have risen by 30%. This increased the carbohydrates in the pollen making it essentially junk food for bees.10
The researchers confirmed the CO2 change in the environment played a role in dropping protein levels through testing published in the Proceedings of the Royal Society B.11
More than 100 past studies have demonstrated the change in atmospheric carbon dioxide reduces the nutritional value of plants, but this study was the first to examine the effects on bees. Mathilde Renard, an agricultural engineer at the Environmental Department of the Paris City Hall, also spoke with the Vice France reporter about the rising pollen counts in Paris.12
She attributes some of the urban pollen to the city’s planting efforts. One of the main strategies Paris is now using is to diversify the species to reduce the pollen concentration for each plant, in the hope of reducing citizens’ allergic response.
Other gardening choices that affect pollen count include preferentially planting male trees.13 Female trees shed more seeds and fruit, which need to be cleaned, but male trees tend to produce more allergic pollen. Some city planners have also chosen species based on their aesthetic value. For instance, birch trees are pretty, but birch tree pollen is one of the largest allergic triggers in the northern hemisphere.14
Oliver attributes the increase in allergies in France to air pollution, saying,15 “It does two things. If you have allergies, it weakens your respiratory tract so you can get ill more easily. It also breaks up the pollen particles in the air, which allows them to penetrate deeper in our respiratory systems.”
The effect of air pollution on your body is insidious. The World Health Organization16 determined 92% of the global population is breathing polluted air. Most people associate air pollution with respiratory conditions since it has a significant impact on your pulmonary health.
However, while the damage to your lungs is significant, it’s important to remember air pollution affects more than your pulmonary system. For example, a study17 published in Environmental Health found a link between living close to busy roads and developing non-Alzheimer’s dementia and Parkinson’s disease.
According to the WHO, by 2016 air pollution was already responsible for respiratory diseases that killed 543,000 children aged 5 years and younger every year.18 Air pollution is also responsible for asthma in 14% of children around the globe. In April 2019, London launched an ultra-low emissions zone in central London in an attempt to reduce emissions by 45%.19
Researchers found that children living in these areas had a reduction in lung capacity by about 5% when pollution rose above legal levels.20 Dr. Ben Barrett commented on studies evaluating the effect air pollution has on pulmonary health in children, saying:21
“Air pollution has been found to restrict lung growth in children. Low lung function in childhood can persist into adulthood and is often associated with other health problems including chronic obstructive lung disease in later life.”
As Oliver commented, air pollution increases the ability of pollen to penetrate your lungs. This can happen through several mechanisms.22 These include the facilitation of pollen release, stimulating the IgE-mediated response and enhancing the expression of allergens within pollen grains.
Grains of pollen do not only carry allergens, but can also elicit allergic responses in individuals who are sensitized. Pollen grains are too large to penetrate deep into the respiratory tract, so symptoms observed with patients who are allergic to pollen are more likely due to particles that are smaller than pollen grains. According to researchers:23
“Interestingly, air pollutants bind to these particles and exacerbate allergic disorders. Moreover, pollen grains release biologically active lipids, which activate immune cells in vitro.”
Researchers postulate that the interaction between air pollution and pollen grains outside the body may increase the amount of allergen released into the environment. This may happen through a variety of mechanisms that researchers have been studying in the lab and with human participants.24
They found that allergic reactions to grass pollen are greater in cities than in rural areas. Several types of air pollutants act “as adjuvants through binding to allergens and stimulating IgE synthesis, resulting in exacerbation of asthma symptoms.”25
The number of people experiencing food allergies is also on the rise. One paper26 published by Yale University27 proposed the rising number may be the result of an exaggerated activation of the system that protects you against eating toxic foods. As many as 8% of children28 have a potentially deadly response to the major eight food allergens, often referred to as the “Big 8.”29
These include milk, eggs, wheat, soybeans, fish, crustacean shellfish, tree nuts and peanuts. Your body uses multiple sensory mechanisms to monitor what you eat, including chemosensory processes in the gut. The Yale University scientists argue that the body has a food quality control system in which an allergic response plays a significant role.
One prevailing theory for this rise in food allergies is living in a too-clean environment. This is also called the hygiene hypothesis, which some scientists have expanded to include processed foods, dishwashing detergent and other environmental chemicals.
In a paper30 written in Clinical and Experimental Immunology, the researchers argue these factors also play a role in disrupting your internal food quality control system.
There is a difference between a food sensitivity, intolerance and a food allergy.31 A true allergy is mediated by the immune system and triggered by a reaction to proteins found in your food or drink. Food intolerances are also called food sensitivities. These are usually an unpleasant gastrointestinal reaction, but the reaction is not mediated by your immune system.
Although most food allergies develop in childhood, it’s not unheard of for adults to develop a food allergy. Data32 gathered from October 2015 to September 2016 suggest 10.8% of adults are allergic to food. Scientists believe this contradicts the long-held belief that most allergies develop in childhood.
In a survey of 40,443 adults, 38.3% had food allergies that sent them to the emergency room and 48% had at least one triggered after the age of 18. Your gut microbiome is vital to the functioning of your immune system, which mediates an allergic response to food. You’ll find suggestions on how to optimize your gut microbiome in “How Your Gut Health Impacts Your Disease Risk.”
One physiological biomarker tied to the risk of seasonal and food allergies is vitamin D. Researchers have found there is both experimental and clinical evidence that vitamin D is linked to allergic rhinitis.33
One double-blind placebo-controlled clinical trial34 evaluated the combined effect of vitamin D supplementation with an antihistamine medication. The study found that the people who received vitamin D and the medication had a significant decrease in their symptoms as compared to those who only received the medication.
The researchers measured vitamin D levels after eight weeks, finding those who received the supplement had a mean serum level of 24 nanograms per milliliter (ng/mL) and the level in the group who did not get vitamin D supplementation was 15 ng/mL.
It is important to note that the researchers did not use the supplement as a basis for concluding the vitamin D had a positive effect, but rather the serum level of vitamin D. The rising level of food allergies also corresponds to the increasing number of people with vitamin D deficiency. Vitamin D plays a significant role in the regulation of IgE, important in the development of food allergies.
The link between vitamin D deficiency, which has almost doubled in just over 10 years in the U.S.,35 and poor regulation of IgE responses, may be a significant factor. Both play a role in the development, severity and course of allergic diseases and may help explain, at least in part, why so many adults are developing food allergies.
Vitamin D deficiency has become so widespread it’s been called a pandemic by a Harvard Medical School researcher.36 The short list of health benefits attributed to vitamin D optimization include improving your immune system, strengthening muscles, bones and teeth and improving your cardiovascular health.
I recommend you get your vitamin D level tested twice each year — once when the level is likely to be at its lowest (midwinter) and once when it’s at its highest (midsummer). Grassroots Health offers vitamin D testing through its D*Action Study and has an online vitamin D calculator you can use to estimate your vitamin D requirements.
To read more about the interaction between vitamin D deficiency and allergies, how to optimize your vitamin D and the synergy between vitamin D3, magnesium, calcium and vitamin K2, see “Vitamin D Deficiency Can Lead to Increased Allergies.”
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